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Albuterol weight loss: The effect of caffeine and albuterol on body composition and metabolic rate

Albuterol Dosage

 

As with nearly all sympathomimetic stimulant based fat loss compounds (and most fat loss compounds in general), the Albuterol doses for the purpose of performance and physique enhancement are generally inflexible. Albuterol is utilized in much the same manner that Clenbuterol or Ephedrine is utilized. Albuterol doses and the protocols in which it is used is actually more similar to Ephedrine than it is to Clenbuterol, although Albuterol’s effects at the cellular level are identical as a beta-2 receptor adrenergic agonist. The manner in which Albuterol doses are administered is closer to Ephedrine in that the half-lives of both compounds are very similar (3 – 6 hours). However, the general manner of use of all stimulants for the purpose of fat loss is normally uniform in the following fashion: a peak dose is selected, which is the maximal daily dose that will be used during the cycle for fat loss. The first several days (normally spanning the first week or two) of use, the dose will be slowly ramped upwards until the final peak dose is achieved, after which the user will remain at the peak dose for the duration of use.

Unlike its close brother Clenbuterol, which is measured in micrograms (mcg), Albuterol doses are measured in milligrams (mg). This is an important point to be remembered, as the various measurements in dosing between not just the many stimulants, but also among the many different performance enhancing drugs, can be very confusing at times (especially for those unfamiliar with chemistry and dosing instructions and measurements).

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Because Albuterol is a beta-2 receptor agonist, like Clenbuterol (and any/all beta-2 receptor agonists), it will over time slowly down regulate the beta-2 receptors[1]. This is especially true under chronic daily use, which is necessary for the fat loss effects that result from Albuterol doses. The beta-2 receptor down regulation will manifest itself as a reduction in effective fat loss over time until no additional fat loss is experienced from Albuterol (which normally takes 2 – 4 weeks to occur, depending on the individual). There are two possible methods of up regulating these receptors, with the first being simple time off the drug (2 weeks minimum or longer). The second method would be through the use of Ketotifen, an anti-histamine drug normally used as an allergy medication, which actively up-regulates these beta-2 receptors[2]. In such an instance, Ketotifen can be utilized at a dose of 2mg every night before bed for 7 days whenever beta-2 receptor down regulation has become significant. This will allow continuous use of Albuterol without the need for breaks. Albuterol, nor any stimulant, should be used chronically at fat loss doses for periods greater than 8 weeks.

Medical Albuterol Dosage

Albuterol is used medically for the treatment and management of asthma, bronchospasm, and to a lesser extent, COPD (chronic obstructive pulmonary disease). For these treatments, Albuterol doses is normally that of 2 – 4mg administered 3 – 4 times daily in tablet/oral format. Each Albuterol dosage is to be spaced evenly apart so as to avoid any unnecessary overlapping build-up of blood plasma levels of the stimulant.

Albuterol Dosage for Fat Loss

Even though Albuterol has demonstrated a greater degree of proven anabolic capability in humans, it is almost exclusively utilized as a fat loss agent. Even so, the Albuterol dosages required for the anabolic effects are the exact same doses required for the fat loss effects as well. Albuterol doses for either goals are typically the same for all three user tiers (beginner, intermediate, and advanced) due to the fact that there is very little inflexibility with the doses required for various effects from a stimulant such as Albuterol. As previously mentioned, Albuterol doses should be slowly ramped upwards until the peak dose is achieved, and the peak optimal dose might be different for different individuals (some individuals might be more sensitive to stimulants than others). This is why the dose can be slowly adjusted upwards, so as to assess tolerance and determine the best effective Albuterol doses.

A peak fat burning Albuterol dose is normally that of 16 – 24mg per day. Being that nearly all Albuterol tablets are dosed at 4mg per tablet, this would in other words be a total of 4 – 8 tablets per day. This peak dose should be separated into evenly spaced administrations throughout the day. For example, if a peak dose of 16mg/day is desired, then the user will generally administer either 8mg twice daily, or 4mg four times daily. An example of these types of Albuterol doses are as follows:

Albuterol use at 24mg/day total (peak dose)
– 8:00am: 8mg of Albuterol
– 12:00pm: 8mg of Albuterol
– 4:00pm: 8mg of Albuterol

Albuterol doses can be split up in even more frequent administrations than the example listed above if the individual desires. It has been mentioned twice already that individuals should ideally slowly ramp the dose upwards at the beginning of use, especially for beginners or individuals who are very sensitive to stimulants. Note that how fast an individual ramps their dose upwards is also a factor that varies between individuals and their personal preference (some might prefer to ramp their Albuterol dosages up once every 3 days, while others might prefer to increase the dose once every 6 days). The following is a general example of ramping up (also known as titrating upwards)

Albuterol doses:

Outline of Proper Ramp-Up Dosing Protocol (for Albuterol at 24mg/day total peak dose)
– Day 1: 8mg of Albuterol
– Day 4: 16mg of Albuterol
– Day 8: 24mg of Albuterol
The user now remains at 24mg/day for the duration of the Albuterol cycle.

Female Albuterol Dosage

The female response to Albuterol is the exact same as the male response, with the only difference being perhaps a variation in the peak dose and/or the slow ramp-up period. Females might exhibit greater sensitivity to Albuterol doses due to the fact that females generally tend to comprise a lower overall body mass and body weight. The same ramp-up protocols should be observed by females, with more attention paid to the individual response and comfort ability. When a comfortable and tolerable Albuterol dosages is achieved, the female can elect to stop at said dose and remain at that peak dose for the duration of use.

Proper Administration and Timing of Albuterol Dosages

Albuterol possesses a half-life of 4 – 6 hours, which necessitates a more frequent multiple-times-per-day dosing schedule, as outlined above. Users will typically spread their Albuterol doses evenly apart throughout the day among 3 – 4 (or more, depending on user preference) administrations. Those who enjoy Albuterol over Clen normally do so because of its shorter half-life, allowing almost all administrations early in the day with little interference of sleeping patterns at night compared to Clenbuterol.

Expectations and Results from Albuterol Dosages

With a caloric deficit and proper training program, Albuterol can increase fat metabolism and aid in the loss of body fat, commonly resulting in an extra 2 – 4 lbs. per month lost from the inclusion of Albuterol alone. With a caloric surplus, Albuterol can indeed exhibit a small but noticeable (but by no means dramatic) increase in strength and muscle mass.

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Albuterol References:

[1] Effect of dietary clenbuterol and cimaterol on muscle composition, beta-adrenergic and androgen receptor concentrations in broiler chickens. Schiavone A, Tarantola M, Perona G, Pagliasso S, Badino P, Odore R, Cuniberti B, Lussiana C. J Anim Physiol Anim Nutr (Berl). 2004 Apr;88(3-4):94-100.

[2]Effects of ketotifen and clenbuterol on beta-adrenergic receptor functions of lymphocytes and on plasma TXB-2 levels of asthmatic patients. Huszar E, Herjavecz I, Boszormenyi-Nagy G, Slapke J, Schreiber J, Debreczeni LA. Z Erkr Atmungsorgane. 1990;175(3):141-6.

Albuterol and Weight Loss: Dosage and Side Effects

One of the fat loss agents I’ve kept in my toolbox for many years is the popular asthma treatment Albuterol.

I featured it in all my books — Metabolic Blowtorch Diet, TOT Bible, Guaranteed Shredded, and Living A Fully Optimized Life — for a good reason:

It’s a powerful fat-burning agent in select bodybuilding circles.

You may balk at this idea as you’ve seen or had direct experience with Albuterol through an inhaler.

But make no mistake about it: If you’re a highly advanced individual who wants to get rid of pockets of stubborn fat and reach single-digit body fat, you’re sorely missing out.

Therefore, this article will be the Jay Campbell treatment on how to use Albuterol for weight loss.

Table of Contents

What Is Albuterol?

Since Albuterol was never intended to be a fat loss drug, I won’t spend too much time on its background and what it was initially intended for.

Albuterol (a.k.a. Salbutamol for non-USA residents) is a bronchodilator (i.e. relaxes/opens up the airways and makes breathing easier) that is primarily used to treat asthma, bronchospasm, and Chronic Obstructive Pulmonary Disease (COPD).

(Source)

It has a long rich history, dating back to the 1960s thanks to pharmaceutical chemist David Jack:

“He appreciated the importance of delivering asthma treatments directly to the lungs by inhalation to produce a more rapid effect and to have fewer systemic side effects than drugs given by mouth.

Working with Roy Brittain, he recognised the advantage of developing selective activators of β2-receptors to avoid cardiovascular side effects from stimulating β1-receptors that were seen with the non-selective inhaled β-agonist isoprenaline.

This led to the discovery of salbutamol (Ventolin), the first selective β2-receptor agonist, which was launched in 1969″

How selective? About 29 times more:

“Salbutamol is a short-acting, selective beta2-adrenergic receptor agonist used in the treatment of asthma and COPD.

It is 29 times more selective for beta2 receptors than beta1 receptors giving it higher specificity for pulmonary beta receptors versus beta1-adrenergic receptors located in the heart. “

This makes a significant difference given the unique locations of each receptor:

Beta1 receptors are located mainly in the heart. When stimulated with a medication, heart rate increases and the heart pumps more blood. 

Beta2 receptors are located in the breathing tubes and in the blood vessels (arteries) that provide  blood to muscles in the arms and legs. When stimulated with a medication, the muscle that wraps around the breathing tubes relaxes allowing more air to go in and out of the lungs.”

As it turns out, there are numerous types of bronchodilator treatments and each of them has its respective place in addressing symptoms of respiratory diseases.

Here’s where Albuterol serves its role as a fast-acting agent:

“Doctors often call short-acting bronchodilators rescue or fast-acting inhalers because they treat symptoms that come on suddenly, such as wheezing, shortness of breath, and chest tightness.

Short-acting bronchodilators work quickly, usually within a few minutes. Although they work fast, the therapeutic effects generally only last 4–5 hours. Short-acting bronchodilators treat sudden symptoms, and people do not need to use them when they are symptom-free.”

Hence the need for asthmatic patients to carry their inhalers everywhere they go… while an agent like Albuterol stops immediate symptoms, long-acting agents like salmeterol keep the airways open for several hours and ideally prevent asthma attacks from happening.

You’ll find numerous brand names for generic Albuterol, with Ventolin being the most common one used in the United States when it received FDA approval in 1980.

So how can we use Albuterol for bodybuilding if it is commonly used as an asthma treatment?

How Does Albuterol Work For Fat Loss?

If you paid attention to the previous section, you may have seen the word “adrenergic” come up:

“[drugs] that mimic or interfere with the functioning of the sympathetic nervous system by affecting the release or action of norepinephrine and epinephrine. These hormones, which are also known as noradrenaline and adrenaline, are secreted by the adrenal gland, hence their association with the term adrenergic.

“…they constrict blood vessels (vasoconstriction), which increases blood pressure, and accelerate the rate and force of contractions of the heart. Adrenergic drugs that produce or inhibit these effects are known as sympathomimetic agents [stimulate the sympathetic nervous system] and sympatholytic agents [inhibit sympathetic nervous system activity], respectively.

And seeing as Albuterol is a central nervous system (CNS) stimulant, this piqued curiosity about its potential role in fat reduction.

There is ample research to support it, some of which I highlight in my book The TOT Bible:

“…Beta-2 receptor agonists are known to accelerate fat burning, especially when they are in a low insulin environment (typically produced during the end of a long fasting window, roughly 14-20 hours in).”

Because Albuterol is a beta-2 receptor agonist, it will slowly downregulate the beta-2 receptors over time441. This means that its fat loss effects are lessened, and eventually fat loss from Albuterol will stall.”

This was back in 2018, and the now-larger body of literature supports the beta-2 receptor as a therapeutic target for the treatment of obesity:

“Individuals with obesity also have lower abundance of beta2-adrenergic receptors in adipose tissue and are less sensitive to catecholamines. In addition, studies in livestock and rodents demonstrate that selective beta2-agonists induce a so-called ‘repartitioning effect’ characterized by muscle accretion and reduced fat deposition.

In humans, beta2-agonists dose-dependently increase resting metabolic rate by 10–50%. And like that observed in other mammals, only a few weeks of treatment with beta2-agonists increases muscle mass and reduces fat mass in young healthy individuals.

Beta2-agonists also exert beneficial effects on body composition when used concomitantly with training and act additively to increase muscle strength and mass during periods with resistance training”

Read closely… those same receptors also happen to be located in our fat cells!

To dive into the mechanism more intimately, here’s how Albuterol triggers fat loss at the cellular level:

“…salbutamol acts by activating adenylate cyclase and, after a sequence of reactions by stimulating the activity of hormone sensitive lipase, which promotes the degradation of triglycerides into free fatty acids and glycerol, providing substrate for beta-oxidation (metabolic pathway that oxidizes the fatty acid into acetyl-CoA).

Furthermore it is also a thermogenic compound (increases the rate of basal metabolism) and anorectic (appetite decreases) which makes it a “fat burner” very efficient. Its effect on fat burning is shown”

We see an increase in fat oxidation and energy expenditure happening with other beta-2 adrenergic agonists, but what about Albuterol?

In one small study with 9 lean male subjects who received Albuterol, the following observations were noted:

  • Energy expenditure increased by 13% [i.e. higher metabolic rate]
  • Fat oxidation increased by 19%
  • Free fatty acid levels in the blood increased by 57%

The last point is especially important as it confirms the fat loss mechanism I just described and allows the fat to be burned off as fuel.

Coupled with raising body temperature and increasing blood flow, you have a super-potent fat loss agent.

Even more so for stubborn fat at low body fat percentages, since it is typically located in “low blood flow” areas of the body like lower abdominal areas for men and glute/ham tie areas for women.

(BTW – this article is an excellent read on the inner workings of stubborn body fat)

It’s no wonder that as early as 2009, people were attempting to create an oral slow-release formulation of Albuterol and make it the next blockbuster drug for obesity!

Scientific Evidence Supporting Albuterol For Fat Loss

Having spoken in a more general matter about drugs like Albuterol, I want to briefly show you the literature for Albuterol and weight loss.

Metabolic Rate

In a randomized, double-blind, crossover study examining 8 healthy men and women (18-50 years old), energy expenditure was almost 5 times higher with 100 mg caffeine and 4 mg albuterol compared to 100 mg caffeine on its own. [NOTE: this website simplifies the study findings into easy-to-read bites]

In a study of twelve lean and young healthy men who engaged in quadriceps exercises, leg oxygen consumption (an indirect measure of metabolic rate) was nearly 2-fold higher for Albuterol compared to placebo prior to exercising, while also being higher after exercising.

Combined with what we learned in the prior section, we have another mechanism to explain the connection between Albuterol and weight loss.

Exercise Performance

In both healthy men and women, the same group of researchers found that short-term or chronic administration of 12 mg Albuterol per day increased maximal anaerobic power, regardless of training status.

In a 2005 study examining Albuterol use (16 mg/day) in weeks 2-3 of a 14-week resistance exercise program, the drug lead to greater strength gains and a higher lean body mass compared to placebo.

A 1997 literature review of performance-enhancing drugs dug up the following studies on beta-2 agonists:

“Martineau et al. [1992] examined the effect of 14 and 21 days of treatment with salbutamol 8mg twice daily on strength in 12 healthy men… there was a significant improvement in strength for the hamstring muscle and quadriceps muscle but not for hand grip muscles… the average strength improvement was about 10 to 15%”

“Caruso et al. [1995] examined the effect of albuterol 16 mg/day or placebo ingested for 6 weeks. Participants were placed on a 9-week isokinetic strength training programme for the knee extensors. Most measures of strength showed a greater increase on the drug than the placebo.”

“Bedi [1988] found that aerosol salbutamol 180 mcg improved cycling sprint time following a 1-hour ride in 3 of the 15 individuals tested. Participants had different levels of training, including highly trained cyclists, triathletes, untrained individuals and recreational athletes. The amount of improvement ranged from 4 to 178 seconds over that of the placebo. If the outliers were discounted, the difference between the placebo and the drug was not significant.”

“Signorile et al. [1992] reported that albuterol 180mcg inhaled resulted in an increase in peak power during sprint cycling in nonathletes.”

In a 2000 study involving eight recreational male athletes, short-term use of oral Albuterol use at 12 mg per day “improved performance during intense submaximal exercise.

And in another 2000 study where nonasthmatic men took 4 mg of oral Albuterol prior to exercising, their mean endurance time and peak torque greatly improved.

Two studies (here and here) did not see any exercise performance improvements in endurance athletes, although they used aerosolized Albuterol at very low dosages… more on that later.

Anabolic (Muscle-Building Properties)

One study involving 65 patients with facioscapulohumeral muscular dystrophy saw muscle volume increases in those who took 16 mg of Albuterol orally per day for 26 weeks.

Albuterol was observed to enhance muscle cell growth during the proliferation stage of skeletal muscle cells, and one experiment conducted on senescent rats saw an increase in skeletal muscle weight for both old and young rats.

In Parkinson’s Disease patients and young boys with Duchenne/Becker muscular dystrophy, 12-16 mg of oral Albuterol per day led to notable increases in lean body mass.

Iron Magazine puts forth a possible explanation for why these observations have taken place:

“Beta-2 adrenoreceptor agonists, such as clenbuterol and albuterol, have been shown to activate cyclic-Adenosine Monophosphate (cAMP). cAMP then activates calpistatin, which inhibits calpain. Calpain degrades protein in muscle tissue. By inhibiting calpain, beta-2 adrenoreceptor agonists have been shown to be anti-catabolic.”

While I would not categorize Albuterol as a muscle-building agent, you can see how theoretical Albuterol weight loss results would lead to more lean tissue preserved.

Blood Glucose Partitioning

In older mice with Type 2 diabetes, chronic Albuterol treatment improved insulin sensitivity and glucose tolerance after 8 weeks of exercise… plus the effects were still there ~5 weeks after exercising!

This same result was also noted in a different study after mice ate a high-fat diet that induced diabetes.

Albuterol Dosage For Weight Loss

After years of using this agent successfully, here is the best Albuterol dose for weight loss:

3-6 mg taken once per day for two weeks straight (ideally in the early morning before fasted cardio), followed by a two-week “off” period.  

This dosage stays the same for me whenever I want to shed body fat, but it does come with some caveats:

Go for the lower dosing range if you’re below 200 pounds, and the higher dosing range if you’re over 200 pounds

Albuterol’s ability to mobilize stubborn fat tissue is best when used during its short half-life of 4-6 hours, hence why I recommend timing it in the morning before fasted cardio and before eating to maximize absorption (additionally, a high-fat diet inflames the airway of asthma patients)

Albuterol also functions best in a low insulin environment, so ideally you want to be 15% body fat or lower when you start using it (fat people have impaired “beta(2)-adrenoceptor-mediated increases in thermogenesis” and “local lipolysis“)

The “two week on, two week off” cycle is recommended because the body quickly develops a tolerance to Albuterol (i.e. Albuterol down regulates the beta-2 receptors and thus its effectiveness decreases over time)

Albuterol can be found as an oral pill, research chemical liquid, or an inhaler… since you need numerous puffs from an inhaler to reach the needed daily dose (each puff has ~100 mcg of Albuterol), the latter two are best. Plus, the oral formulation was most extensively studied in humans for its benefits on metabolic rate and exercise performance. 

You’ll notice on most forums that many people use Albuterol at 2-4 mg per dose for a total of 3-4 doses per day (6-16 mg total per day) — this is FAR beyond what I have ever used, so it’s up to you to experiment and see what works best for you.

Finally… there’s a notable thermogenic effect when Albuterol is combined with coffee and/or nicotine, which may give biohackers an idea in creating an “Albuterol weight loss” stack.

Feel free to experiment with combining to see what gives you the desired effects without the side effects I’m about to mention.

Albuterol Side Effects & Safety Profile

If you follow my dosing and cycling recommendations, the side effects when using Albuterol for fat loss are both mild and rare.

Some of the most commonly reported Albuterol side effects include:

  • Nausea
  • Headache
  • Involuntary shaking of hands/feet
  • Nervousness
  • Muscle aches

Watch out for undesirable drug-drug interactions with Albuterol, such as diuretics, beta-blockers, tricyclic antidepressants, monoamine oxidase inhibitors, and other short-acting bronchodilators.

Two precautions you may want to take are the following, as suggested by other performance enhancement enthusiasts:

  • Ketofin supplementation — 1-2 mg of this anti-histamine drug alongside Albuterol for 7 days reportedly up regulates beta-2 receptors when they are down regulated, allowing for extended use of Albuterol (although you should never use it for longer than 8 weeks)
  • Taurine supplementation — regular Albuterol use reportedly leads to loss of taurine, which may cause excessive and/or severe muscle pumps. 3-5 grams of Taurine per day is recommended to fix this issue.

One of the largest Reddit threads on Albuterol provides a good balance of benefits and risks associated with using the agent for bodybuilding purposes, so I’ll summarize the main takeaways below:

  • Positives
    • Works as promised for fat loss
    • Muscle preservation with a slight anabolic effect
    • “Pre-workout like effect” that translates to more energy during a workout and new PRs
  • Negatives
    • Shakiness / jitters
    • Anxiety attack
    • Slight increase in heart rate

An Important Note For Professional Athletes

The U. S. Anti-Doping Agency (USADA) has very clear rules in place about the use of Albuterol:

“Inhaled albuterol (also called salbutamol): maximum 1,600 micrograms over 24 hours in divided doses, not to exceed 800 micrograms over 12 hours starting from any dose, as long as it is not used in conjunction with a diuretic or masking agent

**Effective January 1, 2022, the daily dosing time interval for albuterol (salbutamol) will be reduced to 600 micrograms over 8 hours (the maximum daily limit remains 1,600 micrograms).

…Use of an oral (swallowed) form of any beta-2 agonist, such as a tablet or syrup, is prohibited at all times and requires an approved TUE [Therapeutic Use Exemption].”

The World Anti-Doping Agency (WADA) has an outright ban on albuterol and other beta-2 agonists, with the following exception:

“Inhaled salbutamol: maximum 1600 micrograms over 24 hours in divided doses not to exceed 600 micrograms over 8 hours starting from any dose”

These rules are in place because the regulators are smart enough to discern asthmatic athletes from the cheaters:

“According to research published in the journal Sports Medicine, this performance-enhancing effect simply doesn’t exist. The authors found that, in 17 of 19 clinical trials involving non-asthmatic competitive athletes, the performance-enhancing effects of inhaled beta2-agonists could not be proved.

All bets are off, however, when an athlete takes albuterol orally or by injection. When administered in this fashion, albuterol has been found to have anabolic properties, which means it can help build muscle like steroids and, according to a 2020 study published in the British Journal of Sports Medicine, it can also boost sprint and strength performance.”

And you best believe they have strict testing requirements:

“[Albuterol was] used by more than 90% of Olympic athletes during the four Olympic Games (2002–2008) when it was necessary to demonstrate that an athlete had asthma or AHR [Airway hyperresponsiveness] to be permitted to use an IBA [Inhaled β2 Agonist] at the Games.

The List states that: ‘the presence in urine of salbutamol in excess of 1000 ng⋅mL−1 is presumed not to be an intended therapeutic use of the substance and will be considered as an AAF’ [Adverse Analytical Finding, which can lead to a doping sanction].

Long story short: DO NOT use Albuterol if you are a competitive athlete of any kind!

Albuterol Vs. Clenbuterol

Before I end this article, I want to make a brief note about the “Albuterol Vs. Clenbuterol” debate and why I will never recommend the latter over the former.

To summarize my stance in one sentence:

Clenbuterol is WORTHLESS unless you want to *POTENTIALLY* damage your heart musculature.

It’s another beta-2 adrenergic receptor agonist just like Albuterol that increases thermogenesis (i.e. fat-burning), but with some big differences:

“Clen has a really long half life (approx 36 hours) and raises your metabolic rate by about 10% per day. On a side note clen does also seem to help increase muscle mass and causes slow twitch muscle fibres to redesign to fast twitch. Clen will allow you to cut harder while maintaining more muscle mass.

Albuterol has a short half life (6 hours) and has the same effects as clen. However, albuterol does seem to be far safer for the heart and has been shown to actually increase the number of “good” lipids (HDLs). This is why albuterol is the drug of choice for copd and asthma patients while clenbuterol got the shaft.

In terms of other side effects, clenbuterol makes you feel like utter shit at high doses (at least for some people) for the day and half of the day after that you take it. While albuterol seems to give more like a high dose caffeine response and you can come off of it closer to bedtime because of the shorter half-life.”

Clenbuterol is far more reported for heart problems such as tachycardia and overall abuse/overuse compared to Albuterol.

Cardiac toxicity manifests even at low doses, and there are numerous reports such as this one of people who show up to the hospital with palpitations short after a “normal” dose of 5mg.

Ironically, due to its superior anabolic properties compared to Albuterol, WADA classifies Clenbuterol as an anabolic agent and not as a beta-2 receptor agonist.

Other nasty side effects of Clenbuterol include decreased strength due to sodium blockage in muscle fibers and sleep disturbance.

Here are some additional articles to read if you’re still second-guessing your decision between Albuterol vs. Clenbuterol:

  • Poison.org goes deeper into Clenbuterol’s numerous heart toxicities
  • SteroidCycle.org and ThinkSteroids.com provide a scientific overview of Clenbuterol, similar to what I’ve done with Albuterol in this article
  • InsideBodybuilding.com and iSteroids.com compare Clenbuterol and Albuterol with additional points not featured in this article

Where To Buy Albuterol

Although you’ll see articles suggesting Albuterol can only be affordably bought on the “dark web”, I’m not going to suggest that.

I have two sources where you can purchase legitimate Albuterol online without breaking the bank…

High Street Pharma and Iron Daddy have both the inhaled version and the oral tablets. 

You’ll have to search for “salbutamol” instead of “albuterol” if you want to find them.

I’m currently in the middle of evaluating vendors for pharma-grade Albuterol in liquid form and will update this article once I’ve chosen someone who meets my high standards of reliability and quality.

Additional Reading Resources For Albuterol

When it comes to how to use Albuterol for weight loss, it is one of the more straightforward drugs I’ve used in my lifetime.

It is highly powerful for its sole purpose of fat loss.

When it is combined with a fully optimized diet and training program, works wonders for anybody who faces a serious plateau in their journey towards a shredded six-pack.

But if you need more information than what I’ve provided you, here are some worthwhile articles to read…

Wikipedia goes deeper into the history and pharmacology of Albuterol, a real treat for the biochemistry nerds.

The Australian Bodybuilding Forum explores the fat loss mechanisms of Albuterol in immaculate detail while also describing the author’s personal experience with using it. 

MuscleChemistry.com has a great overview of everything discussed in this article if you want a quicker read.

As always…

Raise Your Vibration To Optimize Your Love Creation!

PS – For exclusive access to the fat loss protocols I’ve successfully tested and used on elite performers and successful professionals, join The Fully Optimized Health Private Membership Group.

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Chronic cough: 3 most common causes

Periodic cough is normal. Together with it, our body cleanses the lungs of foreign particles and secretions that can cause infection. It is quite another matter when the cough becomes chronic. It is physically exhausting, interferes with sleep, causes dizziness, wheezing, and even urinary incontinence (especially in women). In this article, we will look at where this unpleasant phenomenon comes from and what can be done about it.

When a person coughs for a day or two, they may think they have caught a cold or the flu virus. If he coughs for a week, illnesses such as allergies or bronchitis may come to mind. If the cough “does not let go” for a month, most begin to worry for real.

In order to consider any disorder as chronic, it must disturb the person for at least a certain period of time. Our case is no exception. If you have been coughing continuously for 8 or more weeks, then you really have a chronic cough. This unpleasant phenomenon has many possible causes. We will consider the main ones right now.

According to American experts, in 90% of cases the fault lies with the following disorders:

  1. Postnasal syndrome (or postnasal drip syndrome),
  2. Asthma,
  3. Acid reflux.

These three disorders are responsible for chronic cough in 9 out of 10 people. Less common causes include infections, certain medications, and lung disease. Consider each of the causes of cough in more detail.

Postnasal drip syndrome

Postnasal drip syndrome is diagnosed when nasal discharge drips or runs down the back of the throat. These secretions can irritate the lining of the throat and cause coughing. Most often, postnasal syndrome occurs in people who constantly catch a cold, as well as in allergy sufferers, patients with rhinitis or sinusitis.

Signs of the syndrome:

  • runny nose,
  • sensation as if liquid had collected in the back of the throat,
  • constant desire to clear the throat of mucus.

Some people experience the so-called “silent” postnasal syndrome. In this case, the only complaint of the patient is a constant cough.

Asthma

It is the second most common cause of cough in adults and the #1 cause in children. In addition to the main symptom, asthmatics have wheezing and shortness of breath. However, some suffer from cough asthma – a separate type of disease in which the only pronounced symptom is a dry cough.

Asthma-related coughing may be seasonal or may follow an upper respiratory tract infection. In these cases, the cough may be aggravated by exposure to cold, dry air, certain vapors and aromas.

Gastroesophageal or acid reflux

Acid reflux occurs when acid from the stomach enters the esophagus. Many people with chronic coughs suffer from heartburn or a sour taste in the mouth because of gastroesophageal reflux. However, in some patients with GERD, the only symptom of the disease is a dry cough.

Next, we will look at less common causes:

  • infections,
  • medication,
  • lung disease.

Respiratory tract infections

An upper respiratory tract infection (such as a cold) can cause a cough that “does not let go” for 8 or more weeks. This may be due to post-nasal or airway inflammation due to infection. Many patients with chronic cough due to a respiratory infection respond well to treatment for postnasal or cough asthma.

Sometimes, after a viral infection of the upper respiratory tract, the patient develops bacterial tracheobronchitis or bacterial sinusitis. Almost all patients with bacterial tracheobronchitis have a cough that produces light yellow, dark green, or brown sputum.

Also, almost all patients with bacterial sinusitis have sinus congestion and nasal discharge that drips or runs down the back of the throat. In bacterial sinusitis, sputum has the same color as in tracheobronchitis. If within 10-14 days the patient continues to produce yellow-green mucus, antibiotics may be needed.

ACE inhibitors (angiotensin converting enzyme)

These drugs, which are often prescribed to treat hypertension, cause a persistent dry cough in 20% of patients. For many patients, switching to another drug improves the situation within 1-2 weeks.

Chronic bronchitis

In this disease, the bronchi are constantly inflamed, which causes coughing, sometimes with sputum. In most cases, chronic bronchitis develops in former or current smokers, as well as in people who are constantly exposed to polluted air.

Lung cancer

In fact, very few people with chronic cough have lung cancer. If you smoke, your chances of contracting this deadly disease increase. Serious concern should appear when a smoker coughs up blood. Another cause for concern is if a person continues to cough for more than a month after quitting smoking.

Eosinophilic bronchitis

This is a special type of airway inflammation that can cause chronic coughing. Eosinophilic bronchitis is diagnosed when a breath test to detect asthma is negative, but microscopic examination of sputum reveals cells called eosinophils.

How to cure a cough?

Treatment should be directed at the underlying cause, not the symptom itself. In most cases, it takes some time to select the most effective method of treatment. By analyzing which cough remedy worked best, the therapist can figure out the exact cause of the persistent cough.

On the other hand, in very many patients the cause is hidden not in one, but in several violations at once. In this case, coughing can be eliminated only if the treatment is directed simultaneously to all pathologies.

Depending on the underlying cause, the therapist may prescribe different medications.

Postnasal syndrome

Postnasal syndrome is relieved by decongestants, nasal or oral antihistamines, nasal glucocorticoids, or sprays containing ipratropium. In this case, the choice of specific drugs for the treatment of cough will depend on the medical history and symptoms. The following are specific examples:

  • If postnasal syndrome appeared after a cold

In this case, the patient is prescribed antihistamines, which are taken in the form of tablets – chlorpheniramine or clemastine. Decongestants are used to relieve nasal congestion and therefore reduce the flow of mucus into the throat.

  • If the postnasal syndrome is due to allergies

In this case, the doctor prescribes nasal glucocorticoids. They help reduce inflammation in the nose and soothe allergic coughs. Oral antihistamines such as loratadine, fexofenadine, and cetirizine are also effective in allergic postnasal syndrome. Unlike previous generation antihistamines (chlorpheniramine, diphenhydramine), new drugs are less likely to cause drowsiness.

Nasal antihistamine sprays, such as azelastine, can also relieve postnasal drip.

All of these cough medicines (nasal glucocorticoids, oral or nasal antihistamines) can be taken alone or in combination.

Cough asthma

If the cause is asthma, the patient is prescribed a standard treatment regimen. It necessarily includes an inhaled glucocorticoid – fluticasone, budesonide or beclomethasone. If the patient is wheezing or short of breath, the doctor will prescribe an inhaled bronchodilator (albuterol). The glucocorticoid reduces the inflammation of the airways, and the bronchodilator “opens” them.

Acid reflux

Treatment of cough caused by reflux should primarily be based on lifestyle changes. Experts recommend:

  • Avoid foods that increase acid reflux: fatty and spicy foods, soda, chocolate, sour juices, garlic, tomatoes, and citrus fruits. You should also limit alcohol consumption,
  • Do not eat 2-3 hours before bedtime,
  • Sleep with the upper body elevated by 15-20 cm, placing the bars under the legs of the bed,
  • Lose weight if you have extra pounds,
  • Quit smoking.

Doctors sometimes prescribe proton pump inhibitors – omeprazole, esomeprazole, lansoprazole. These drugs reduce the production of acid in the stomach. It will take 8 or more weeks of medication to treat the cough in this case.

Eosinophilic bronchitis

For the treatment of this disease, inhaled glucocorticoids already familiar to you are prescribed – budesonide or fluticasone. These drugs reduce inflammation in the airways.

For which symptoms should you see a doctor as soon as possible?

According to scientists from Harvard University, if symptoms from the list below appear along with a persistent cough, you should run to the doctor:

  • Shortness of breath,
  • Unexplained weight loss,
  • Weakness and fatigue,
  • Loss of appetite,
  • Heavy night sweats,
  • Chest pain,
  • Wheezing or wheezing.
  • If you have a chronic cough, don’t wait until the above symptoms appear. It’s better to see a therapist.

    Sources:

    1. Patient education: Chronic cough in adults (Beyond the Basics), UpToDate,
    2. That nagging cough, Harvard University,
    3. 8 Reasons Your Cough Is Not Improving, WebMD.

    National Doping League. What NFL players sit on

    First & Goal looks into what illegal drugs American football players use and why they get away with it.

    Every time you watch the NFL, you are surprised by the size and physical strength of the players. Football requires specific skills combined with incredible athleticism. Receivers and running backs run 40 yards in 4.4 seconds, jump 100 cm, and all this with 100 kilograms of pure muscle mass. Defensive linemen often weigh over 130kg and yet have elite speed and movement on the field. From time to time you read the statements of a player who decided to gain 10-15 kg of pure muscle mass in the off-season. You ask yourself: how is this possible?

    02 STEROID PROHIBITION

    Everyone knows that steroids were widely used in the NFL in the 1970s and 80s, right up to the official ban in 1987. Many famous players openly admitted to using anabolic steroids because it was legal. When the league finally introduced the ban and testing, players received a four-game suspension (a quarter of the regular season) for the first violation, an eight-game suspension for the second, and a year for the third. After that, the players had to apply for reinstatement to the league commissioner.

    STIM PROHIBITED TODAY

    NFL internal policy is to test for banned stimulants year-round. Intensive testing begins during the training camp because all the players are in the same place. During pre-season, regular season and playoff games, ten players from each playing team are tested weekly.

    Testing continues during the off-season: contracted players may be tested up to six times. Each player is subjected to this procedure at least once a year. At the same time, players who fail the university test less than two years before participating in the NFL Draft are subject to a special, tighter control, which involves more frequent testing for at least two years.

    WHAT THE PLAYERS ARE SAYING

    Steroid use peaked in the 1980s, when 20.3% of players reportedly did so. In 2009, 9% of anonymously polled former NFL players admitted to using steroids. The percentage of steroid use is especially high among the linemen of attack (16.3%) and defense (14.8%).

    Two reservations must be made here. Firstly, although the survey is anonymous, it is designed for the honesty of the players. Secondly, in addition to steroids, there are other illegal drugs. The answer to this question depends on whether the interviewed football players understand the word “steroid” in a broad or narrow sense. Therefore, it can be assumed that the actual figures for the use of illicit stimulants are higher.

    The list of drugs banned by the league is quite long and includes familiar names. The problem is that there are loopholes in the testing system, and there are exceptions to the rules. “VITAMIN”

    T”

    Toradol
    not included in the list of prohibited drugs,
    but it deserves to be singled out. Famous
    among players nicknamed T-train, toradol
    is a very powerful non-steroidal
    pain reliever and anti-inflammatory
    means. Theoretically so strong
    painkillers should be given
    doctor’s prescription. In reality, many
    players simply use toradol before
    match so as not to feel pain.
    The drug has become popular
    at 1995 when the Houston Chronicle wrote
    article about the Houston Medical Headquarters
    Oilers.” According to a 2002 survey, 28 out of
    30 games used toradol on game days.
    One player spoke about his experience
    Bleacher Report:

    “I
    got hurt and they shoved me
    pills in my mouth when they bandaged me.
    That was the attitude: “You will play,
    no matter what happens”.

    Scale
    Toradol epidemics are no secret to anyone.
    In 2016, former offensive lineman
    Ravens Eugene Monroe wrote an article
    in the portal
    Players Tribune on
    topic of painkiller abuse
    in the NFL and potential uses
    marijuana instead of toradol.

    HORMONE
    ROSTA

    Testing
    on growth hormone (HGH) in the NFL began only
    in 2014. Surprisingly, so far
    one player was not caught in this violation.
    According to former
    nfl players,
    10 to 40 percent of athletes in the NFL
    use growth hormone. Independent
    denied policy administrator
    stimpacks nfl doctor John Lombardo
    chalks it up to limitation
    tests:

    “Think,
    difficulty with hormone testing
    growth is related to the sensitivity of the test
    and a short detection window. Eat
    another test that is being developed
    with a large detection window, – from 5 to
    8 days. When this test is applied,
    the situation will change. Short window –
    difficult problem in all sports.”

    Window
    NFL test detection for synthetic
    growth hormone is 24 to 48 hours.
    Given that players have 24 hours to
    sampling and blood tests on match days
    banned, it is not surprising that so far
    so far no player has been caught
    synthetic growth hormone. More
    effective test developed by WADA,
    should raise the detection window to
    three weeks.

    IGF-1

    Less
    famous brother of growth hormone, IGF-1
    (insulin-like growth factor) helps
    recover injured players
    tendons, build muscle and become
    stronger and faster. IGF-1 is produced
    in response to growth hormone and is needed,
    for growth hormone to have an effect on muscles
    and fabric. This drug is included in the spray
    from deer antlers, who allegedly used
    famous ray lewis for tear healing
    biceps to recover in time
    to the Super Bowl. isoform tests,
    used by the league, not enough
    effective to detect IGF-1.

    ERYTROPOETIN,
    EPHEDRINE AND ALBUTEROL

    Erythropoietin
    contributes to the formation of red
    blood cells, which are
    carriers of oxygen throughout the body. A drug
    widely used by cyclists
    and difficult to detect, especially if
    used in microdoses. EPO test
    expensive and has a very small window
    detection (24 hours). ephedrine used
    for weight loss, banned in the NFL. Like
    airway-opening albuterol
    (exception for asthmatics). Marvelous,
    but a fairly large percentage
    athletes asthma and, accordingly,
    they can legally consume the drug.

    REFERENCE
    FROM THE DOCTOR TO ADDERALL

    Adderall
    not so famous in Russia, but in the USA it
    every second knows. This drug
    usually prescribed for the treatment of the syndrome
    attention deficit hyperactivity disorder
    (ADHD) in children. psychotropic stimulant,
    amphetamine-based, increases
    mental and physical abilities.
    For people who do not have ADHD, the drug has
    stimulating effect, increasing attention
    and focus. Dr. Leah Lagos
    notes the following reasons for abuse
    adderall:

    “Athletes
    often used to combat
    fatigue. It’s like drinking 100 cups of coffee.
    They can use it during
    training camp when their bodies
    especially exhausted, and before the games to
    get a boost. These are the two most common
    reasons for use.

    Adderall
    included in the list of prohibited drugs
    NFL. But many players have permission
    from doctors to his therapeutic
    usage. In the US, more than 6% of children
    being treated for ADHD, while
    number of diagnoses
    has grown by almost 50% over the past six
    years. If you wish, get help from
    doctor is not difficult. Such
    exception (Therapeutic Use Exemption) allows
    legally use prohibited
    a drug. American gymnast Simon
    Biles also has permission from doctors
    to use adderall.

    Star
    Seahawks cornerback Richard Sherman supposedly
    suspected of consuming adderall,
    but in the end he managed to win the appeal.
    In an interview Sherman openly
    announced
    total use of adderall in
    NFL:

    “Approximately
    half the league accepts Adderall, and the NFL
    it needs to be resolved.”

    IN
    during the season the players spend at the base
    teams eight hours a day looking at
    video recordings, participating in meetings,
    working out in the gym, exercising.
    When the players come home they have to
    watch another three hours of video cuts of games.
    This combination of physical and mental
    loads affect the ability
    players to assimilate information during
    weeks and quickly respond to what is happening
    during the game. For many adderall –
    daily necessity.

    TESTING
    NFL INEFFICIENT

    Victor
    Conte,
    founder of the BALCO laboratory, formerly
    at the center of a doping scandal around
    famous athletes, pretty tough
    speaks of NFL:

    “This
    one hundred percent propaganda. If the NFL
    wanted to catch players who
    dope, they would test
    differently. The league’s policy is ineffective.”

    Apart from
    short majority detection window
    league tests, negative samples are kept
    only 90 days. For comparison, samples taken
    USADA for Olympic testing,
    kept for ten years. Not enough
    cheat test today, new forms
    testing in the future may reveal
    doping that was not detected before
    this. WADA has already created a biological passport for all
    athletes collecting urine and blood tests
    for many years. The introduction of such
    systems in the NFL are unimaginable.

    CONCLUSION

    WITH
    progress in testing and expansion
    banned drugs list players
    switching from anabolic steroids
    to substances that are difficult to detect,
    and microdoses.

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