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Diet controlled type 2 diabetes: The prevention and control the type-2 diabetes by changing lifestyle and dietary pattern

The prevention and control the type-2 diabetes by changing lifestyle and dietary pattern

1. Wild S, Roglic G, Green A, Sicree R, King H. Global prevalence of diabetes: Estimates for the year 2000 and projections for 2030. Diabetes Care. 2004;27:1047–53. [PubMed] [Google Scholar]

2. Giugliano D, Esposito K. Mediterranean diet and metabolic diseases. Curr Opin Lipidol. 2008;19:63–8. [PubMed] [Google Scholar]

3. Martinez-Gonzalez MA, de la Fuente-Arrillaga C, Nunez-Cordoba JM, Basterra-Gortari FJ, Beunza JJ, Vazquez Z, et al. Adherence to Mediterranean diet and risk of developing diabetes: Prospective cohort study. BMJ. 2008;336:1348–51. [PMC free article] [PubMed] [Google Scholar]

4. Sanchez-Tainta A, Estruch R, Bullo M, Corella D, Gomez-Gracia E, Fiol M. Adherence to a Mediterranean-type diet and reduced prevalence of clustered cardiovascular risk factors in a cohort of 3,204 high-risk patients. Eur J Cardiovasc Prev Rehabil. 2008;15:589–93. [PubMed] [Google Scholar]

5. Panagiotakos DB, Pitsavos C, Chrysohoou C, Stefanadis C. The epidemiology of Type 2 diabetes mellitus in Greek adults: The ATTICA study. Diabet Med. 2005;22:1581–8. [PubMed] [Google Scholar]

6. Geneva: WHO; 1999. World Health Organization. Definition, Diagnosis and Classification of Diabetes Mellitus and Its Complications. Part 1. Report of WHO consultation. [Google Scholar]

7. Report of the WHO consultation on obesity. Geneva: WHO; 1998. Prevention and Management of the Global Epidemic of Obesity. [Google Scholar]

8. Otuyelu F. Diabetic diet for the Nigerian. Niger Med Pract. 1982;3:48–51. [Google Scholar]

9. Fadupin GT, Keshinro OO. Factors influencincing dietary compliance and glycaemic control in adult diabetic patients in Nigeria. Diabetes Int. 2001;11:59–61. [Google Scholar]

10. Fadupin GT, Keshinro OO, Sule ON. Dietary recommendations: Example of advice given to diabetic patients in Nigeria. Diabetes Int. 2000;10:68–70. [Google Scholar]

11. Sofi F, Innocenti G, Dini C, Masi L, Battistini NC, Brandi ML, et al. Low adherence of a clinically healthy Italian population to nutritional recommendations for primary prevention of chronic diseases. Nutr Metab Cardiovasc Dis. 2006;16:436–44. [PubMed] [Google Scholar]

12. Kastorini CM, Panagiotakos DB. Mediterranean diet and diabetes prevention: Myth or fact? World J Diabetes. 2010;1:65–7. [PMC free article] [PubMed] [Google Scholar]

13. Colditz GA, Willett WC, Rotnitzky A, Manson JE. Weight gain as a risk factor for clinical diabetes mellitus in women. Ann Intern Med. 1995;122:481–6. [PubMed] [Google Scholar]

14. Feskins EJ, van Dam RM. Dietary fat and the etiology of type 2 diabetes: An epidemiological perspective. Nutr Metab Cardiovasc Dis. 1999;9:87–95. [PubMed] [Google Scholar]

15. Bergnman RN, Ader L. Free fatty acids and the pathogenesis of type 2 diabetes mellitus. Trends Endocrinol Metab. 2001;3(suppl):S11–9. [Google Scholar]

16. Panagiotakos DB, Polystipioti A, Papairakleous N, Polychronopoulos E. Long-term adoption of a Mediterranean diet is associated with a better health status in elderly people; a cross-sectional survey in Cyprus. Asia Pac J Clin Nutr. 2007;16:331–7. [PubMed] [Google Scholar]

17. Panagiotakos DB, Tzima N, Pitsavos C, Chrysohoou C, Zampelas A, Toussoulis D, et al. The association between adherence to the Mediterranean diet and fasting indices of glucose homoeostasis: The Attica Study. J Am Coll Nutr. 2007;26:32–8. [PubMed] [Google Scholar]

18. American Diabetes Association. Standards of medical care in diabetes-2010. Diabetes Care. 2010;33(suppl 1):S11–61. [PMC free article] [PubMed] [Google Scholar]

19. American Diabetes Association: Evidence-based nutrition principles and recommendations for the treatment and prevention of diabetes and related complications (Position Statement) Diabetes Care. 2003;26(Suppl 1):S51–61. [PubMed] [Google Scholar]

20. Barnard ND, Scialli AR, Turner-McGrievy G, Lanou AJ, Glass J. The effects of a lowfat, plant-based dietary intervention on body weight, metabolism, and insulin sensitivity. Am J Med. 2005;118:991–7. [PubMed] [Google Scholar]

21. American Diabetes Association. Standards of Medical Care in Diabetes-2008. Diabetes Care. 2008;31:S12–S54. [PubMed] [Google Scholar]

22. Barnard ND, Scialli AR, Turner-McGrievy GM, Lanou AJ. Acceptability of a low-fat vegan diet compares favorably to a step II diet in a randomized, controlled trial. J Cardiopulm Rehabil. 2004;24:229–35. [PubMed] [Google Scholar]

23. Barnard RJ, Jung T, Inkeles SB. Diet and exercise in the treatment of NIDDM: The need for early emphasis. Diabetes Care. 1994;17:1–4. [PubMed] [Google Scholar]

24. Wieland H, Seidel D. A simple specific method for precipitation of low density lipoproteins. J Lipid Res. 1983;24:904–9. [PubMed] [Google Scholar]

25. Yang W, Lu J, Weng J, Jia W, Ji L, Xiao J, et al. Prevalence of diabetes among men and women in China. N Engl J Med. 2010;362:1090–101. [PubMed] [Google Scholar]

26. Holt SH, Brand Miller JC, Petocz P, Farmakaladis E. A satiety index of common foods. Eur J Clin Nutr. 1995;49:675–90. [PubMed] [Google Scholar]

27. Nicholson AS, Sklar M, Barnard ND, Gore S, Sullivan R, Browning S. Toward improved management of NIDDM: A randomized, controlled, pilot intervention using a low-fat, vegetarian diet. Prev Med. 1999;29:87–91. [PubMed] [Google Scholar]

28. UK Prospective Diabetes Group. Intensive bloodglucose control with sulphonylureas and insulin compared with conventional treatment and risk of complications in patients with type 2 diabetes (UKPDS 33) Lancet. 1998;352:837–53. [PubMed] [Google Scholar]

29. Wolf AM, Conaway MR, Crowther JQ, Hazen KY, L Nadler J, Oneida B, et al. Translating lifestyle intervention to practice in obese patients with type 2 diabetes: Improving control with activity and nutrition (ICAN) study. Diabetes Care. 2004;27:1570–6. [PubMed] [Google Scholar]

30. The Diabetes Control and Complications Trial Research Group. The effect of intensive treatment of diabetes on the development and progression of long-term complications in insulin-dependent diabetes mellitus. N Engl J Med. 1993;329:977–86. [PubMed] [Google Scholar]

31. The International Expert Committee. International Expert Committee report on the role of the A1C assay in the diagnosis of diabetes. Diabetes Care. 2009;32:1327–34. [PMC free article] [PubMed] [Google Scholar]

32. Sinitskaya N, Gourmelen S, Schuster-Klein C, Guardiola-Lemaitre B, Pevet P, Challet E. Increasing fat-to-carbohydrate ratio in a high-fat diet prevents the development of obesity but not a prediabetic state in rats. Clin Sci. 2007;113:417–25. [PubMed] [Google Scholar]

33. Sjostrom CD, Peltonen M, Wedel H, Sjostrom L. Differentiated long-term effects of intentional weight loss on diabetes and hypertension. Hypertension. 2000;36:20–5. [PubMed] [Google Scholar]

34. Schroder H. Protective mechanisms of the Mediterranean diet in obesity and type 2 diabetes. J Nutr Biochem. 2007;18:149–60. [PubMed] [Google Scholar]

35. Selvin E, Crainiceanu CM, Brancati FL, Coresh J. Short-term variability in measures of glycemia and implications for the classification of diabetes. Arch Intern Med. 2007;167:1545–51. [PubMed] [Google Scholar]

36. Shai I, Schwarzfuchs D, Henkin Y, Shahar DR, Witkow S, Greenberg I, et al. Weight loss with a lowcarbohydrate, Mediterranean, or low-fat diet. N Engl J Med. 2008;359:229–41. [PubMed] [Google Scholar]

37. Lindeberg S, Jönsson T, Granfeldt Y, Borgstrand E, Soffman J, Sjöström K, et al. A Palaeolithic diet improves glucose tolerance more than a Mediterranean-like diet in individuals with ischaemic heart disease. Diabetologia. 2007;50:1795–807. [PubMed] [Google Scholar]

38. Lovejoy JC, Windhauser MM, Rood JC, de la Bretonne JA. Effect of a controlled high-fat versus low-fat diet on insulin sensitivity and leptin levels in African-American and Caucasian women. Metabolism. 1998;47:1520–4. [PubMed] [Google Scholar]

39. American Diabetes Association. Clinical practice recommendations-2007. Diabetes Management in Correctional Institutions: Diabetes Care. 2007;30(Supp 1):S77–84. [PubMed] [Google Scholar]

40. American Diabetes Association. Diagnosis and classification of diabetes mellitus. Diabetes Care. 2010;33(Suppl 1):S62–9. [PMC free article] [PubMed] [Google Scholar]

41. Bouchard C, Tremblay A, LeBlanc C, Lortie G, Savard R, Theriault G. A method to assess energy expenditure in children and adults. Am J Clin Nutr. 1983;37:461–7. [PubMed] [Google Scholar]

42. Drewnowski A. Energy density, palatability, and satiety: Implications for weight control. Nutr Rev. 1998;56:347–53. [PubMed] [Google Scholar]

43. Duncan KH, Bacon JA, Weinsier RL. The effects of high and low energy density diets on satiety, energy intake, and eating time of obese and nonobese subjects. Am J Clin Nutr. 1983;37:763–7. [PubMed] [Google Scholar]

44. Eaton SB, Eaton SB, Konner MJ, Shostak M. An evolutionary perspective enhances understanding of human nutritional requirements. J Nutr. 1996;126:1732–40. [PubMed] [Google Scholar]

45. Esposito K, Maiorino MI, Di Palo C, Giugliano D. Adherence to a Mediterranean diet and glycaemic control in Type 2 diabetes mellitus. Diabet Med. 2009;26:900–7. [PubMed] [Google Scholar]

46. Franz MJ, Splett PL, Monk A, Barry B, McClain K, Weaver T, et al. Cost-effectiveness of medical nutrition therapy provided by dietitians for persons with non-insulin-dependent diabetes mellitus. J Am Diet Assoc. 1995;95:1018–24. [PubMed] [Google Scholar]

47. Friedewald WT, Levy RI, Fredrickson DS. Estimation of the concentration of lowdensity lipoprotein cholesterol in plasma without use of the preparative ultracentrifuge. Clin Chem. 1972;18:499–502. [PubMed] [Google Scholar]

48. Garg A, Bantle JP, Henry RR, Coulston AM, Griver KA, Raatz SK, et al. Effects of varying carbohydrate content of the diet in patients with non-insulin-dependent diabetes mellitus. JAMA. 1994;271:1421–8. [PubMed] [Google Scholar]

49. Garg A. High-monosaturated fat diets for patients with diabetes mellitus: A meta-analysis. Am J Clin Nutr. 1998;67:577S–82. [PubMed] [Google Scholar]

50. Howarth NC, Saltzman E, Roberts SB. Dietary fiber and weight regulation (Review) Nutr Rev. 2001;59:129–39. [PubMed] [Google Scholar]

51. Pérez-Jiménez F, López-Miranda J, Pinillos MD, Gómez P, Paz-Rojas E, Montilla P, et al. A Mediterranean and a high-carbohydrate diet improve glucose metabolism in healthy young persons. Diabetologia. 2001;44:2038–43. [PubMed] [Google Scholar]

52. Schakel SF, Sievert YA, Buzzard IM. Sources of data for developing and maintaining a nutrient database. J Am Diet Assoc. 1988;88:1268–71. [PubMed] [Google Scholar]

53. Simopoulos AP. The Mediterranean diets: What is so special about the diet of Greece? The scientific evidence. J Nutr. 2001;131:3065S–73. [PubMed] [Google Scholar]

54. Wilson DH, Bogacz JP, Forsythe CM, Turk PJ, Lane TL, Gates RC, et al. Fully automated assay of glycohemoglobin with the Abbott IMx analyzer: Novel approaches for separation and detection. Clin Chem. 1993;39:2090–7. [PubMed] [Google Scholar]

55. Weiss R, Dufour S, Taksali SE, Tamborlane WV, Petersen KF, Bonadonna RC, et al. Prediabetes in obese youth: A syndrome of impaired glucose tolerance, severe insulin resistance, and altered myocellular and abdominal fat partitioning. Lancet. 2003;362:951–7. [PMC free article] [PubMed] [Google Scholar]

56. Sparks LM, Xie H, Koza RA, Mynatt R, Hulver MW, Bray GA, et al. A highfat diet coordinately downregulates genes required for mitochondrial oxidative phosphorylation in skeletal muscle. Diabetes. 2005;54:1926–33. [PubMed] [Google Scholar]

57. Centers for Disease Control and Prevention. Transmission of hepatitis B virus among persons undergoing blood glucose monitoring in long-term-care facilities–Mississippi, North Carolina, and Los Angeles County, California, 2003–2004. MMWR Morb Mortal Wkly Rep. 2005;54:220–3. [PubMed] [Google Scholar]

58. Estruch R, Martínez-González MA, Corella D, Salas-Salvadó J, Ruiz-Gutiérrez V, Covas MI, et al. Effects of a Mediterranean-style diet on cardiovascular risk factors: A randomized trial. Ann Intern Med. 2006;145:1–11. [PubMed] [Google Scholar]

59. Federal Bureau of Prisons. Washington, DC: Federal Bureau of Prisons; 2007. Federal Bureau of Prisons, Clinical practice guidelines: Preventive health care. [Google Scholar]

60. Goff LM, Bell JD, So PW, Dornhorst A, Frost GS. Veganism and its relationship with insulin resistance and intramyocellular lipid. Eur J Clin Nutr. 2005;59:291–8. [PubMed] [Google Scholar]

61. Atlanta, GA: US Department of Health and Human Services, Centers for Disease Control and Prevention; 2008. [Last accessed on 2010 May 18]. Centers for Disease Control and Prevention. National Diabetes Fact Sheet: General Information and National Estimates on Diabetes in the United States 2007. [Google Scholar]

62. Field AE, Willett WC, Lissner L, Colditz GA. Dietary fat and weight gain among women in the Nurses’ Health Study. Obesity. 2007;15:967–6. [PubMed] [Google Scholar]

63. Ford ES, Giles WH, Dietz WH. Prevalence of the metabolic syndrome among US adults. Findings fromthe Third National Health and Nutrition Survey. JAMA. 2001;287:356–9. [PubMed] [Google Scholar]

64. Fraser GE. Diet, Life Expectancy, and Chronic Disease. Oxford UK: Oxford University Press; 2003. Vegetarianism and obesity, hypertension, diabetes, and arthritis; pp. 129–48. [Google Scholar]

65. Trichopoulou A. Mediterranean diet: The past and the present. Nutr Metab Cardiovasc Dis. 2001;11:1–4. [PubMed] [Google Scholar]

66. US Preventive Services Task Force. Screening for type 2 diabetes mellitus in adults: US Preventive Services Task Force Recommendation Statement. Ann Intern Med. 2008;148:846–54. [PubMed] [Google Scholar]

67. Nathan DM, Kuenen J, Borg R, Zheng H, Schoenfeld D, Heine RJ for the A1C-derived Average Glucose (ADAG) Study Group. Translating the A1C assay into estimated average glucose values. Diabetes Care. 2008;31:1473–8. [PMC free article] [PubMed] [Google Scholar]

68. Pan XR, Li GW, Hu YH, Wang JX, Yang WY, An ZX, et al. Effects of diet and exercise in preventing NIDDM in people with impaired glucose tolerance: The Da Qing IGT and Diabetes Study. Diabetes Care. 1997;20:537–44. [PubMed] [Google Scholar]

69. Patel RB, Burke TF. Urbanization: An emerging humanitarian disaster. N Engl J Med. 2009;361:741–3. [PubMed] [Google Scholar]

70. Petersen KF, Dufour S, Befroy D, Garcia R, Shulman GI. Impaired mitochondrial activity in the insulin-resistant offspring of patients with type 2 diabetes. N Engl J Med. 2004;350:664–71. [PMC free article] [PubMed] [Google Scholar]

71. Qi L, Hu FB, Hu G. Genes, environment, and interactions in prevention of type 2 diabetes: A focus on physical activity and lifestyle changes. Curr Mol Med. 2008;8:519–32. [PubMed] [Google Scholar]

72. Abelson R. New York: The New York Times; 2010. An insurer’s new approach to diabetes; p. 14. [Google Scholar]

73. Hu FB. Dietary fat and meat intake in relation to risk of type 2 diabetes in men. Diabetes Care. 2002;25:417–24. [PubMed] [Google Scholar]

74. Hua NW, Stoohs RA, Facchini FS. Low iron status and enhanced insulin sensitivity in lacto-ovo vegetarians. Br J Nutr. 2001;86:515–9. [PubMed] [Google Scholar]

75. Kendall A, Levitsky DA, Strupp BJ, Lissner L. Weight loss on a low-fat diet: Consequence of the imprecision of the control of food intake in humans. Am J Clin Nutr. 1991;53:1124–9. [PubMed] [Google Scholar]

76. Kleges RC, Kleges LM, Haddock CK, Eck LH. A longitudinal analysis of the impact of dietary intake and physical activity on weight change in adults. Am J Clin Nutr. 1992;55:818–22. [PubMed] [Google Scholar]

77. Rolls BJ. The role of energy density in the over consumption of fat. J Nutr. 2000;130:268S–71. [PubMed] [Google Scholar]

78. Zammit VA, Waterman IJ, Topping D, McKay G. Insulin stimulation of hepatic triacylglycerol secretion and the etiology of insulin resistance. J Nutr. 2001;131:2074–7. [PubMed] [Google Scholar]

79. Hossain P, Kawar B, El Nahas M. Obesity and diabetes in the developing world-a growing challenge. N Engl J Med. 2007;356:213–5. [PubMed] [Google Scholar]

80. Ackerman RT, Finch EA, Brizendine E, Zhou H, Marrero DG Translating the Diabetes Prevention Program into the community. The Deploy Pilot Study. Am J Prev Med. 2008;35:357–63. [PMC free article] [PubMed] [Google Scholar]

81. Allain CC, Poon LS, Chan CS, Richmond W, Fu PC. Enzymatic determination of total serum cholesterol. Clin Chem. 1974;20:470–5. [PubMed] [Google Scholar]

82. Gaede PH, Lund-Andersen H, Parving HH, Pedersen O. Effect of a multifactorial intervention on mortality in type 2 diabetes. N Engl J Med. 2008;358:580–91. [PubMed] [Google Scholar]

83. Guldstrand M, Adamson U, Ahrxen B. Improved β-cell function after standardized weight reduction in severely obese subjects. Am J Physiol Endocrinol Metab. 2003;284:E557–65. [PubMed] [Google Scholar]

84. Haber GB, Heaton KW, Murphy D, Burroughs LF. Depletion and disruption of dietary fibre: Effects on satiety, plasma-glucose, and serum-insulin. Lancet. 1977;2:679–82. [PubMed] [Google Scholar]

85. Laitinen JH, Ahola IE, Sarkkinen ES, Winberg RL, Harmaakorpi-Iivonen PA, Uusitupa MI. Impact of intensified dietary therapy on energy and nutrient intakes and fatty acid composition of serum lipids in patients with recently diagnosed noninsulin- dependent diabetes mellitus. J Am Diet Assoc. 1993;93:276–83. [PubMed] [Google Scholar]

86. Mattson JS, Cerutis RD. Diabetes mellitus: A review of the literature and dental implications. Compend Contin Educ Dent. 2001;22:757–70. [PubMed] [Google Scholar]

87. Mokdad AH. The continuing epidemics of obesity and diabetes in the United States. JAMA. 2001;286:1195–200. [PubMed] [Google Scholar]

88. Morgan SA, O’Dea K, Sinclair AJ. A low-fat diet supplemented with monounsaturated fat results in less HDL-C lowering than a very-low-fat diet. J Am Diet Assoc. 1997;97:151–6. [PubMed] [Google Scholar]

89. Jenkins DJ, Kendall CW, Marchie A, Faulkner DA, Wong JM, de Souza R, et al. Effects of a dietary portfolio on cholesterol-lowering foods vs lovastatin on serum lipids and C-reactive protein. JAMA. 2003;290:502–10. [PubMed] [Google Scholar]

90. Jenkins DJ, Kendall CW, Marchie A, Jenkins AL, Augustin LS, Ludwig DS, et al. Type 2 diabetes and the vegetarian diet. Am J Clin Nutr. 2003;78:610S–6. [PubMed] [Google Scholar]

91. Krentz AJ, Bailey CJ. Oral antidiabetic agents: Current role in type 2 diabetes mellitus (Review) Drugs. 2005;65:385–411. [PubMed] [Google Scholar]

92. Asif M. The role of fruits, vegetables, and spices in diabetes. Int J Nutr Pharmacol Neurol Dis. 2011;1:26–34. [Google Scholar]

93. Roberts CK, Vaziri ND, Barnard RJ. Effect of diet and exercise intervention on blood pressure, insulin, oxidative stress, and nitric oxide availability. Circulation. 2002;106:2530–2. [PubMed] [Google Scholar]

94. Stubbs RJ, Johnstone AM, Harbron CG, Reid C. Covert manipulation of energy density of high carbohydrate diet in ‘pseudo free-living’ humans. Int J Obes. 1998;22:885–92. [PubMed] [Google Scholar]

95. Tremblay A. Nutritional determinants of the insulin resistance syndrome. Int J Obes. 1995;19(suppl):S60–5. [PubMed] [Google Scholar]

96. Vrieze A, Holleman F, Zoetendal EG, de Vos WM, Hoekstra JB, Nieuwdorp M. The environment within: How gut microbiota may influence metabolism and body composition. Diabetologia. 2010;53:606–13. [PMC free article] [PubMed] [Google Scholar]

97. Wieland H, Seidel D. A simple specific method for precipitation of low density lipoproteins. J Lipid Res. 1983;24:904–9. [PubMed] [Google Scholar]

98. Yang W, Lu J, Weng J, Jia W, Ji L, Xiao J, et al. Prevalence of diabetes among men and women in China. N Engl J Med. 2010;362:1090–101. [PubMed] [Google Scholar]

99. Holt SH, Brand Miller JC, Petocz P, Farmakaladis E. A satiety index of common foods. Eur J Clin Nutr. 1995;49:675–90. [PubMed] [Google Scholar]

Controlling type 2 diabetes: With and without medication

Many people may wonder how to control type 2 diabetes without medications. A healthy diet and lifestyle could help people manage type 2 diabetes and other aspects of their health.

To help people keep blood sugar — blood glucose — within a healthy range, the American Heart Association (AHA) recommends:

  • engaging in weight management
  • eating a nutritious diet
  • getting regular exercise
  • stopping smoking
  • reducing stress

If making dietary and lifestyle changes do not help maintain a healthy blood sugar level, doctors may advise a person to take medications. However, if someone receives a diabetes diagnosis as an older adult and their blood sugar is only mildly elevated, medications may not be necessary.

In this article, we examine how to control type 2 diabetes without medication. We also look at the causes of type 2 diabetes and when people may need medication to manage their condition.

A 2020 study reports that healthy lifestyle practices could benefit people with type 2 diabetes or risk factors for the condition. Such measures may delay or prevent its development, as well as treat or potentially put it into remission. This method of controlling blood sugar can be so effective that the study’s authors call it lifestyle medicine.

The following healthy lifestyle practices may help reduce blood sugar levels:

1. Pursue weight management

In people with overweight or obesity, significant weight loss may reduce blood sugar from the diabetic to the nondiabetic range.

Two ways to manage weight are eating a healthy, balanced diet and engaging in regular exercise. The key to weight loss involves consuming fewer calories than the body uses for activities and physiological processes.

2. Eat a healthy diet

A healthy diet consists of eating nutritious foods in appropriate portion sizes while avoiding or limiting non-nutritious foods.

Foods for people to eat may include:

  • whole grains, such as oatmeal, brown rice, and whole grain bread
  • fruits and vegetables
  • non-fried fish that is high in omega-3 fatty acids, such as salmon and lake trout
  • lean meat, such as sirloin and white meat from chicken or turkey
  • nontropical vegetable oils, such as olive oil
  • unsalted nuts and seeds
  • legumes, such as beans and peas
  • low fat dairy products

Foods and ingredients for people to limit may include:

  • sugary foods and beverages, such as candy, cakes, jelly, honey, sodas, sweet tea, fruit drinks, and concentrated fruit juices
  • sweet food additives, such as high fructose corn syrup, dextrose, maltose, fructose, and sucrose
  • processed and fatty meats, such as bacon, hot dogs, and fatty cuts of beef and pork
  • salty foods
  • partially hydrogenated and trans fat foods, such as shortening, hard margarine, microwave popcorn, frozen pizzas, desserts, and coffee creamer
  • saturated fat, such as foods containing palm oil or coconut oil

The American Diabetes Association (ADA) recommends a diet similar to the Mediterranean diet, which focuses on:

  • fruits
  • vegetables
  • whole grains
  • nuts
  • olive oil
  • fatty fish

A 2020 review notes that following this eating plan improves blood sugar control.

3. Get regular exercise

Exercise promotes blood sugar management and burns calories, which contributes to weight loss. Physical activity also increases insulin sensitivity, which helps blood sugar to enter the cells from the bloodstream.

People should aim to get 30 minutes of moderate physical activity per day on most days, totaling at least 150 minutes each week. Experts classify a brisk walk as moderate exercise. Alternatively, 75 minutes per week of vigorous aerobic activity is equally beneficial.

4. Stop smoking

Doctors advise people to stop smoking to help blood sugar control for several reasons. Smokers have a 30–40% higher risk of developing diabetes than nonsmokers. Smoking also makes exercise more challenging.

Smoking also raises blood sugar temporarily, which poses an additional challenge in maintaining nondiabetic blood sugar levels. This increases the likelihood of a person developing complications of diabetes, such as kidney disease and nerve damage.

5. Manage stress

Research in 2019 suggests that although stress does not cause type 2 diabetes, it can worsen it. Stress stimulates the release of hormones that interfere with the body’s blood sugar regulation. It also makes a person more likely to engage in practices that make it harder to manage blood sugar, such as overeating and smoking.

One way to reduce stress involves taking a break from electronics and spending time in nature.

According to 2020 research, a person only needs medication if lifestyle practices do not put blood sugar levels in the nondiabetic range.

A doctor’s recommendation for medication for someone with type 2 diabetes may depend partly on their age when they receive a diagnosis. While many older adults with the condition have slightly higher blood sugar levels, this rarely causes problems.

On the other hand, doctors may prescribe medications to people who receive a diagnosis by the age 40 or 50. Even slightly elevated blood sugar levels can eventually lead to health problems, such as damage to nerves or blood vessels. Such damage may result in complications, such as kidney disease. The purpose of medications is to delay or prevent the harmful effects of diabetes.

According to the ADA, type 2 diabetes is progressive, making it more difficult to manage over time. Improvements in medical care enable people with the condition to live longer. However, despite the advancements, type 2 diabetes may reduce life expectancy by up to 10 years.

The effects of lifestyle practices alone on type 2 diabetes have not undergone extensive research, limiting statistics on the results of such interventions. However, a 2018 clinical trial examined the outcomes of a weight management program on 306 individuals with type 2 diabetes. After 12 months, the authors found about half the individuals who participated in the program went into remission.

Researchers cannot quantify the exact improvement that each healthy lifestyle practice may bring at this point in research. However, the outlook for people with type 2 diabetes who have a healthy lifestyle is better than those who do not.

Type 2 diabetes is a condition that involves high blood glucose or blood sugar.

The pancreas makes insulin, a hormone that enables cells to take glucose from the bloodstream for energy. In type 2 diabetes, the cells do not respond normally to insulin, called insulin resistance. As a result, the pancreas makes more insulin in an attempt to get glucose inside the cells.

After some time, the pancreas cannot keep up, and blood sugar increases, which leads to prediabetes and diabetes.

Symptoms frequently develop over several years, including:

  • tiredness
  • increased thirst and urination
  • blurry vision
  • increased hunger
  • slow healing of sores
  • numbness or tingling in hands or feet
  • weight loss without trying
  • dry skin
  • more infections than usual

Experts advise people interested in learning how to control type 2 diabetes without medications to adopt a healthy lifestyle.

Significant weight loss can help control blood sugar levels in some people. Two ways to pursue weight management involve people eating a healthy, balanced diet and getting regular exercise.

Good nutrition is vital for a person with type 2 diabetes. Some evidence suggests that a nutritious eating plan, such as the Mediterranean diet, may help control blood sugar in ways other than weight loss.

Type 2 Diabetes Diet: Do’s and Don’ts

November 14 is World Diabetes Day. One of the important points in the treatment of diabetes is diet. We tell you which foods are allowed for type 2 diabetes, and which ones are better to forget about.

A person diagnosed with type 2 diabetes manages to maintain normal blood glucose levels only with the help of a special diet. But in order to achieve excellent results in compensating for the disease, it is important to know what should not be eaten with diabetes, which foods should be limited as much as possible, and which can be consumed without restrictions. It is helpful for people with diabetes to keep a food diary to help control their diet.

What you can eat:

  • Animal and vegetable proteins (meat, poultry, fish, eggs, cheese, cottage cheese, legumes).
  • Fats of animal and vegetable origin (butter, sour cream and cottage cheese with a high fat content, vegetable oils, nuts).
  • Only slow carbohydrates (cereals, vegetables, greens).
  • Spices, spices, herbs.

What not to eat:

Products that aggravate carbohydrate metabolism disorders. These include:

  • Foods with a high glycemic index (flour, sugar, chocolate, white rice, potatoes, cooked beets, carrots, etc.). Carbohydrates in them are quickly broken down and cause sharp jumps in blood glucose.
  • Products containing lactose. It is recommended to limit the consumption of milk and liquid dairy products to 100 ml per day.
  • Fructose reduces the sensitivity of cells to insulin, which is already reduced in type 2 diabetes. Therefore, fruits, dried fruits, honey should be limited in the diet (15 g of fructose per day). For example, very sweet fruits (bananas, pears, grapes) should be excluded. It is better to give preference to berries. And use this group of products as a dessert, do not use it as an independent dish. Many snack on fruit – this is wrong.

Type 2 Diabetes Food Chart

Type of permitted product



Limited: apricots, cherry plums, grapefruits, oranges,
lingonberries, cranberries, peaches, apples, plums, sea buckthorn,
red currants, cherries, gooseberries


Cucumbers, broccoli, green peas, cauliflower,
lettuce, greens, zucchini, green pepper, mushrooms

Dairy products

Low-fat milk, cottage cheese, fermented milk products,
some low-fat cheese


Soybeans, beans, peas, lentils, buckwheat, oatmeal,
pearl barley, occasional pasta

Meat and fish

Lean beef, turkey, chicken, rabbit, white
fish species


Special diabetic products only
and very limited


Unsweetened tea, coffee, compotes, herbal decoctions

Bakery products

Whole wheat bread


Eggs, vegetable oils

Weekly menu for type 2 diabetes mellitus

This is what an approximate diet for a type 2 diabetic looks like. Doctors recommend not to make a menu on your own, but to seek advice from nutritionists.


Breakfast: buckwheat porridge

Lunch: beef stew with vegetables

Snack: baked apple with cottage cheese

Dinner: pink salmon on vegetable pillow


Breakfast: barley porridge

Lunch: onion soup or beef with vegetables

Snack: cabbage salad with apple

Dinner: lean fish


Breakfast: millet porridge with pumpkin

Lunch: chicken fillets and cereals to choose from allowed

Snack: cottage cheese with berries

Dinner: lean fish or turkey with stewed vegetables


Breakfast: scrambled eggs (no more than two eggs per serving)

Lunch: chicken with grits

Afternoon snack: cheesecakes

Dinner: lean beef and vegetables


Breakfast: cheesecakes

Lunch: white fish fillets in oven with ovo shami

Snack: cabbage salad

Dinner: turkey


Breakfast: any porridge from the list of allowed

Lunch: mashed vegetable soup in a slow cooker

Snack: kiwi smoothie

Dinner: chicken or turkey with vegetables 9 0003


Breakfast: oatmeal

Lunch: chicken soup

Afternoon snack: salad with celery and walnuts

Dinner: lean fish fillet with vegetables

Results of diet 9 0003

Proper nutrition for type 2 diabetes helps to lose weight, and thus thereby reduce the insulin resistance of cells, in other words, susceptibility to insulin.

Dieting for Type 2 Diabetes may seem tedious at first, but it will eventually become a way of life.

Diabetes is incurable, but it can be controlled and lead a full life. In patients who follow the rules and recommendations, blood sugar normalizes, the general condition of the body improves and immunity increases. A person can also get rid of extra pounds and all sorts of problems associated with diabetes.

You need to remember the main rule – food should be varied. It is important that with food a person receives vitamins and other useful substances necessary for the life of the body.

Natalya Belyaeva, dietitian:

“Type 2 diabetes belongs to the NCD group of diseases – non-communicable diseases characterized by a long duration and slow progression. By the way, in addition to type 2 diabetes, this group also includes cardiovascular diseases, oncological diseases and chronic respiratory diseases.

Nutrition as a component of a proper lifestyle in type 2 diabetes mellitus, of course, must be organized in accordance with the existing metabolic disorders – be complete, contain all macro- and micronutrients. It is important with nutrition not only to correct the disturbed carbohydrate metabolism in such patients, but also to prevent the formation of other problems “ .

According to the site “HEALTHY EATING” (https://healthy-food.rf)

menu for a week, what you can and cannot eat, reviews of nutritionists 9 0001

Benefits of diet for diabetes

Diabetes mellitus is an endocrine disease in which carbohydrate metabolism is disturbed. The pancreas produces the hormone insulin, which is responsible for lowering blood sugar levels. A lack of insulin causes a state of hyperglycemia – an increased concentration of glucose in the blood.

The most common form of the disease is type 2 diabetes , which most often occurs in people over forty years of age, mainly in women. The development of the disease contributes to reduced physical activity, obesity, genetic predisposition, menopause and subsequent hormonal disorders. Cells do not respond to insulin and do not absorb glucose from the blood, although the amount of the hormone is still normal. The concentration of sugar rises, in response to insulin, even more is produced. Cells react even weaker to an increased dose of the hormone, insulin resistance occurs.

Since cells do not receive enough glucose, despite a large amount of it in the blood, patients often experience hunger and weakness, eat even more carbohydrates, which aggravates the situation and leads to obesity. Such conditions are well corrected by proper nutrition, that is, a decrease in the amount of carbohydrates consumed in order to reduce the load on the pancreas and not cause a rise in blood sugar.

Type 1 diabetes occurs in children and young adults and is caused by a pathological condition of the pancreas that produces little or no insulin. Such patients are forced to artificially introduce this hormone for life.

There is also gestational diabetes in pregnant women . Hormones released during childbearing block insulin, as a result of which sugar rises. Usually, after childbirth, the condition returns to normal on its own.

The main problem in any type of diabetes is a pathologically high level of glucose in the blood and carbohydrate starvation of cells that receive less sugar. To reduce sudden spikes in sugar, insulin and reduce the load on the pancreas, reduce the amount of carbohydrates consumed, focusing on “slow carbohydrates” that give a feeling of satiety. Fatty foods are also excluded, since they increase the secretion of pancreatic enzymes and lead to the same increased load on the organ.

Disadvantages of the diet for diabetes mellitus

The main disadvantage is dietary restrictions. A large number of products are excluded, the method of their preparation becomes as sparing and dietary as possible, which may seem tasteless to those who are accustomed to otherwise. Also, fractional frequent meals for a uniform intake of carbohydrates cause certain difficulties during the day, when it is difficult to find the right snack that does not contain sugars.

In severe diabetes, the glycemic index of each food item should be monitored, excluding anything where the sugar concentration is too high.

Many artificial sweeteners to create a sweet taste without raising blood glucose levels are harmful to the body. Natural substitutes are recommended, for example, stevia extract, erythritol. However, natural preparations can also cause sugar rises. According to numerous studies, fructose also contributes to obesity, although to a lesser extent than sucrose. Before and after meals, sugar is usually measured with special devices.

Diabetes Food Chart

Modern approaches to therapy include reducing the carbohydrate load in the diet to 5-7%, and increasing the consumption of vegetable fats. As a result of such a correction, the body receives a full range of useful micro and macro elements, without a large amount of carbohydrates, thereby reducing the carbohydrate load and the need for insulin preparations. Of course, in type 1 diabetes, it is impossible to completely give up insulin, but the development of type 2 diabetes can be halted and even reversed.

Patients are prescribed Diet No. 9 or its variations. The amount of carbohydrates is adjusted by the doctor depending on the degree of the disease, the weight of the patient.

In the past, diabetics were advised to completely eliminate easily digestible, fast carbohydrates. But today, WHO prioritizes such a concept as the quality of human life, so doctors are gradually abandoning radical formulations such as “this is strictly forbidden to you”, “forget about sweets”, etc.

Studies have shown that such restrictions are stressful for a person, and stress can lead to breakdowns, depression, so that life with continuous “no” will not be a joy. In addition, we still need sugar, because carbohydrates are energy for life. You cannot replace them with proteins and fats.

Therefore, the diet for diabetics is based on the glycemic index of all products. In fact, this index reflects the rate at which sugar from them is absorbed into the blood. In foods with a high index – jam, cookies, cakes – the absorption rate is high. Products with a low index – cereals, fruits – are lower. The slower the rate of absorption, the easier it is for the pancreas to cope with the load. So, foods with a GI of up to 55 are broken down gradually and almost do not cause an increase in sugar.

The essence of the diet is to consume more foods with a low glycemic index during the day and to dose meals.

— Both white bread and black bread are carbohydrates. The use of both increases blood sugar levels, the difference is only in speed. The first has a little more, and the second has a little less, so we say that black bread is preferable for diabetics, ”endocrinologists explain.

What you can eat

Cooking increases the GI of food, so it is desirable to eat a lot of food raw or not boil food.

Type of permitted product Products
Fruit grapefruits, oranges, lingonberries, cranberries, peaches, apples, plums, sea buckthorn, red currants, cherries, gooseberries
Vegetables Cucumbers, broccoli, green peas, cauliflower, lettuce, herbs, zucchini, green peppers, mushrooms
Dairy products Low-fat milk, cottage cheese, fermented milk products, some low-fat cheese
Cereals Soybeans, beans, peas, lentils, buckwheat, oatmeal, barley, occasionally pasta
Meat and fish Lean beef, turkey, chicken, rabbit, white fish
Sweet Only special diabetic products and extremely limited
Drinking Unsweetened tea, coffee, compotes, herbal decoctions
Bakery products Whole wheat bread
Miscellaneous Eggs, vegetable oils

What not to eat

Doctors are now trying not to operate with such formulations. Firstly, with type 1 diabetes, you can eat almost everything, but you have to count bread units and inject insulin. Secondly, even with type 2 diabetes, in which diet plays a role, you can eat varied, but you should try to limit fast carbohydrates, fatty foods and other foods from the table below.

However, even here there are nuances. Take at least the same fatty foods. It is high in calories, and obesity often goes hand in hand with type 2 diabetes, so the number of calories for such people needs to be reduced. But at the same time, according to doctors, fats limit the absorption of sugar, so mashed potatoes on water will raise sugar levels faster than fried potatoes in oil.

And, of course, the degree of restriction of certain products depends on human activity. If you play sports, you need more carbohydrates than knowledge workers.

Prohibited product Food
Sweet ice cream, syrups
Vegetables Severely restrict potatoes, carrots and beets
Cereals Corn, semolina, rice
Bakery products Fresh pastries from fine flour
Meat and fish Lamb, fatty roast meat, fatty red fish
Drinks Juices, sodas, alcohol ready meals, sausages, dumplings, french fries, etc.
Fruit Grapes, watermelon, melon, sweet and canned fruit
Miscellaneous Pickles, marinades, spicy 049

Weekly menu for diabetes diet

The main goal is to prevent large fluctuations in blood glucose. For this, it is assumed that all food is divided into small parts that are consumed all day. At least 5 meals. As snacks, you can use cottage cheese, kefir, low-fat cheese, nuts.

In the nutrition menu for diabetics, the amount of carbohydrates should be evenly distributed, which is determined by the doctor for each patient separately. In type 1 diabetes, when the patient is dependent on artificial insulin administration, a low-carbohydrate diet may reduce the dose of the required drug, since blood sugar will be lower.

Reducing the amount of carbohydrates in the diet, which increase blood sugar, reduces the need for medication. The use of the same dosages as before the transition to a low-carbohydrate diet can cause hypoglycemia. It is necessary to regularly measure blood sugar during the beginning of this diet and adjust the dosage of medications accordingly. This must be done under medical supervision! Specific products, their quantity, dependence on taking drugs, are chosen only by the doctor for each patient separately. This menu for a week with diabetes can be considered as a guide and an example of a patient’s diet.

By deliberately reducing sugar and starch in the diet, blood glucose stabilizes and the level of insulin, the hormone that controls body fat, decreases. This speeds up fat burning and increases the feeling of satiety after eating.

Day 1

Breakfast: Omlet with vegetables (tomatoes, cucumbers, herbs)
Lunch: vegetable oil, boiled chicken breast and avocado
Dinner: chicken and broccoli casserole

Day 2

Breakfast: 2 boiled eggs, boiled asparagus
Lunch: fish grilled or baked with vegetables, lettuce
90 262 Dinner: beef steak with baked mushrooms

Day 3

Breakfast: whole wheat spaghetti with zucchini
Lunch: turkey in cream sauce with vegetables, leaf lettuce
Dinner: avocado salad before, bacon and cheese

Day 4

Breakfast: bulgur porridge with blueberries and lemon zest
Lunch: cod with vegetables, stewed lentils
Dinner: chicken in garlic sauce, crumbly buckwheat

Day 5

Breakfast: boiled eggs, toasted whole grain bread
Lunch: bean stew, diet beef
Dinner: vegetable salad with herbs, cheese slice

Day 6

Breakfast: low-fat cottage cheese
Lunch: salad with vegetables, egg and tuna
Dinner : braised rabbit or white fish, boiled cauliflower

Day 7

9 0541 Breakfast: barley porridge with diluted milk
Lunch: Steamed beef patties, Diet mushroom soup
Dinner : Baked fish with pesto and vegetables


Controlled intake of sugar reduces insulin resistance. Fractional nutrition allows you to avoid sharp jumps in sugar: both lowering and increasing, harmful to the pancreas and the body as a whole. In insulin-dependent diabetes, diet is necessary to match the dose of the drug and approximately the same amount of carbohydrates. If they are deficient, the dose of insulin may be too high and hypoglycemia will occur, and vice versa.

Nutritionists Reviews

— Proper, balanced nutrition is of paramount importance in type 1 and type 2 diabetes, but to call it a diet, in the narrow sense of the word, would not be correct. Although reducing carbohydrates and fats in the diet contributes to weight loss, excess of which often affects type 2 diabetics. It is important to change the lifestyle itself, food habits. Since the main problem in both type 1 and type 2 diabetes is due to the fact that the body does not properly use the glucose that comes with food, the main focus of a properly designed nutrition plan should be on foods that include starch and simple sugars – their should be avoided.