Treatment, home management, and prevention

Blood clotting is a vital process that prevents excessive bleeding following an injury to a blood vessel. However, a blood clot can sometimes form inside a blood vessel that has not sustained any damage.

Some clots may also fail to dissolve fully once an injury has healed. These clots can travel through the circulatory system and may end up restricting blood supply to a vital organ. These types of blood clot are very serious and require urgent treatment.

In this article, we outline the various treatments for blood clots and provide tips on the prevention and long-term management of blood clotting disorders.

Share on PinterestA person with DVT may benefit from wearing compression stockings.

A doctor will set out a tailored treatment plan to help people manage their condition and prevent further blood clots.

Home management will depend on the type and severity of the blood clot, as well as which medications a person is taking for it.

The plan may involve the doctor referring a person to a team of specialists, who are likely to include a cardiologist, hematologist, and neurologist.

Compression stockings may be beneficial for people recovering from DVT. These stockings help prevent blood from pooling and clotting in the lower leg.

Taking regular walks and elevating the affected leg above the hip can also help by increasing blood flow to the heart.

People who are taking medications should schedule regular blood checks to ensure that their blood does not become too thin or thick.

According to the American Society of Hematology, blood clots are one of the most preventable types of blood condition.

Some individuals may have an increased genetic risk of developing blood clots. People should tell their doctor if they have a family history of blood clotting disorders.

The doctor may recommend regular checkups to help detect disorders at an early stage.

The following lifestyle factors can also lower a person’s risk of developing blood clots:

• wearing loose fitting clothes, especially on the lower body
• wearing compression stockings
• quitting smoking, if applicable
• drinking plenty of fluids
• eating less salt
• exercising regularly
• maintaining a healthy weight
• changing position often, especially on long journeys
• standing or sitting for no longer than an hour at a time
• avoiding crossing the legs
• avoiding activities that may bump and knock the legs
• raising the legs above the level of the heart when lying down

The table below shows the symptoms that may occur with blood clots in different parts of the body:

A DVT is a blood clot that forms within the deep veins in the arms and legs.

Sometimes, a DVT may dislodge and travel through the circulatory system toward the lungs or brain. It can then cause serious complications, including:

Pulmonary embolism

A PE is a blood clot that becomes embedded in the lung tissue.

A PE blocks some of the blood flow to the lung, which causes the heart to pump harder to try to fulfill the body’s oxygen requirements.

The added strain on the circulatory system can result in heart failure.

Cerebral embolism and stroke

Sometimes, a blood clot may enter and block a vessel that supplies blood to the brain. This type of blood clot is called a cerebral embolism (CE).

Without sufficient blood supply, the brain cells in the affected area become starved of oxygen and die. This condition is called an ischemic stroke.

Strokes are serious and potentially life threatening. People who receive treatment within the first 3 hours of having a stroke are less likely to experience lasting disability.

Renal vein thrombosis

A renal vein thrombosis (RVT) is a blood clot in the renal vein, which draws blood away from the kidney. Most cases of RVT improve over time and do not cause lasting kidney damage.

However, RVT may sometimes result in acute renal failure. Acute renal failure is when impaired kidney function leads to a buildup of toxic waste products in the blood.

Share on PinterestA doctor will recommend early treatment of DVT to prevent further complications.

People who experience the symptoms of a DVT should make an urgent appointment with a doctor, especially if they have a family history of blood clotting disorders. Early treatment of a DVT can prevent further complications.

A blood clot elsewhere in the body is a medical emergency. A person should phone 911 or the local emergency number immediately if they are experiencing symptoms of a stroke, pulmonary embolism, renal vein thrombosis, or another heart-related condition.

Blood clots are serious and potentially life threatening in some cases. There are many different treatment options for blood clots, depending on their location and severity.

Early diagnosis and treatment can help prevent further complications and improve a person’s outlook.

Blood Clot Treatments | Anticoagulants

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• What is a Blood Clot?
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• Oral Anticoagulants
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• Long-term Management of Clotting Disorders
• Warfarin Interactions with Vitamin K
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Common Anticoagulant Medications

Once a patient is diagnosed with a blood clot, blood thinners, or anticoagulants, are used to reduce the ability of the blood to clot. Depending on the type and location of the blood clot, a person will often be treated with a blood thinner for approximately three to six months, but sometimes the person will need to take the treatment for the rest of their life.

Learn more about long-term management of clotting disorders

The total length of treatment depends on if there is also:

Identified underlying risk factors, such as an abnormality in a coagulation factor, also affect the length of treatment.

The length of blood-thinning therapy is based on clinically proven evidence as well as individual factors. Blood thinners can cause serious bleeding and should only be taken under the direction of a healthcare provider.

COMMON BLOOD THINNER MEDICATIONS

Oral Anticoagulants

Learn more about oral anticoagulants

Injectable Anticoagulants

• Enoxaparin (Lovenox)

• Dalteparin (Fragmin)

• Fondaparinux (Arixtra)

Learn more about injectable anticoagulants

Intravenous Anticoagulants

Learn more about intravenous anticoagulants

Please note that this is not a comprehensive list of all anticoagulants.

Thrombosis Treatment

• Oral Anticoagulants
• Injectable Anticoagulants
• Intravenous Anticoagulants

• Long-Term Management of Clotting Disorders
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• Warfarin Dietary Tips

• What is a Blood Clot?
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Thrombosis. What is this? How to treat thrombosis?

Recently, thrombosis, thrombophlebitis and thromboembolism are often diagnosed. However, not everyone understands correctly and can at least in general terms explain what thrombosis is. Meanwhile, this condition happened to everyone. And certainly you were told about this phenomenon at a school biology lesson.

So, thrombosis is the formation of a blood clot (thrombus) inside the vascular bed (venous, arterial or capillary). It is caused by a number of factors, the most common of which is trauma with damage to the vessel, inside which a blood clot forms. What is a thrombus for? A blood clot (thrombus), closing the damaged vessel, prevents liquid blood from flowing out. This mechanism is very important for the body, without it we would die from bleeding as a result of even the smallest cut.

What is a thrombus? This is a blood clot formed by plasma proteins (a liquid fraction of blood) that polymerize (create long protein chains) and in which some blood cells, in particular platelets, “get stuck” or stick. Thus, thrombosis is one of the most important evolutionary factors that allowed organisms with a circulatory system to survive.

Thrombus (blood clot) under a microscope

Unfortunately, any mechanism of our body fails from time to time. Excess formation of blood clots or a violation of the reverse mechanism – thrombolysis (and this is the body’s own mechanism that can dissolve blood clots) can lead to thrombosis, which interferes with the vascular system to circulate blood. In addition, the formed clot can break away from the attachment site and move to other parts of the circulatory system, causing a sudden blockage of healthy vessels.

Arterial thrombosis

When it comes to the arterial system, arterial thrombosis can lead to catastrophic consequences. We are talking about the occurrence of acute starvation (ischemia, heart attack), which develops very quickly (usually within minutes, less often – hours) and leads to irreversible consequences – the death of an organ or tissue, and sometimes the whole organism. Our organs and tissues continuously consume nutrients and oxygen. As soon as the delivery of these substances is reduced to a critical level, cells die off, the work of the organ is disrupted, which inevitably affects the activity of the whole organism. The latter reacts to such “starvation” (ischemia) with severe pain and other signals, depending on the function and location of the organ (or limb). It is very difficult to miss such a problem.

Thrombosis in the arterial system, if there is no obvious injury to the vessel, is overwhelmingly associated with atherosclerosis. Let me remind you that atherosclerosis is the deposition of cholesterol plaques in the wall of the arteries with the formation of a narrowing of the vessel. The arterial bed experiences significant stress associated with the need to constantly pump blood and nourish organs and tissues with nutrients and oxygen.

Normal arterial pressure on the vessels of the forearm (standard blood pressure measuring point) is 120 to 80 mmHg. Art. By the way, do you know why blood pressure is exactly 120 over 80? The fact is that the upper pressure is formed during the contraction of the heart (or rather the left ventricle), which pushes the blood into the main, most important and largest vessel – the aorta. After the heart contracts and pushes blood into the aorta, the aortic valve closes so that the blood does not flow back into the heart, but is distributed to the numerous aortic branches, nourishing the organs and tissues. The pressure in the aorta at the peak of contraction reaches 120 mm Hg. Art. Further, the heart “relaxes” (although modern physiologists have proven that this is also an energy-intensive process), taking a portion of blood from the lungs, and the pressure in the aorta begins to decrease to 80 mm Hg. Art. This is how lower blood pressure is formed. Then the process is repeated. The heart is responsible for maintaining blood pressure (as the main pump), as well as the “capacity” of the channel (that is, the amount of free space where blood can be pumped). The capacity of the vascular bed is regulated by two factors: the diameter of the lumen of the arteries and the total volume of blood in the human body.

Let’s return to atherosclerosis and arterial thrombosis. As we have already found out, atherosclerosis is a disease associated with the deposition of cholesterol in the vessel wall and leading to the formation of plaques (growths) on the arterial wall. By themselves, plaques begin to pose a danger when they narrow the lumen of the vessel so much that it leads to starvation (ischemia) of the organ or tissue associated with the vessel. In the process of growth, the plaque can completely block the diseased vessel or burst due to increased blood pressure. In this case, the same mechanism of thrombus formation is triggered as in case of damage to the vessel, causing thrombosis.

Atherosclerotic plaque inside the artery

Features of arterial thrombosis

A feature of arterial thrombosis is the indispensable participation in it of special blood cells – platelets. They stick to each other, forming a strong blood clot (thrombus), capable of withstanding significant pressure fluctuations inherent in the arterial bed, but, unfortunately, in parallel causing ischemia of the nourished organ.

The second feature of atherosclerosis is that this disease develops with age. Cholesterol plaques are gradually deposited in the arteries. Usually the disease begins to manifest itself after 40-50 years. Women are “protected” by female sex hormones, preventing the deposition of cholesterol. Diabetes, smoking, unhealthy diet (fatty, salty foods), high blood pressure accelerate the growth of plaques. Therefore, a patient with atherosclerosis should receive drugs that act against arterial thrombosis.

Aspirin for prevention

The most studied drug is acetylsalicylic acid (included in the preparations are aspirin, cardiomagnyl, thromboASS ). It was synthesized more than 100 years ago, however, it has been used for about 30 years as a prevention of arterial thrombosis. The thing is that aspirin blocks the enzyme cyclooxygenase-1, which is involved in the aggregation (gluing) of platelets. As a result, even if an atherosclerotic plaque ruptures, thrombosis does not form or has a less pronounced course. Therefore, aspirin is prescribed specifically for age-related patients with signs of atherosclerosis and a high risk of arterial thrombosis as a result of rupture of a cholesterol plaque.

Other more modern and powerful drugs that block the development of arterial thrombosis are clopidogrel ( Plavix ) and ticagrelor ( Brilinta ). They are prescribed to patients with a high risk of arterial thrombosis (after the installation of stents in the artery, coronary artery bypass grafting).

If the doctor suspects that the patient has atherosclerosis (or the disease is proven by research), aspirin or an aspirin-containing drug is prescribed for prevention and a mandatory low-cholesterol diet is recommended, as well as checking cholesterol and sugar levels every six months. In the event of sharp pains in the problematic organ or limb, the patient is immediately hospitalized in the vascular departments of clinics.

Venous thrombosis

Modern studies have shown that venous thrombosis is based on a fundamentally different mechanism of blood clot formation. In addition, the development of venous thrombosis (unlike arterial) does not always lead to a critical lesion of the organ or tissue in which it develops. In this regard, a number of thromboses are asymptomatic or generally asymptomatic until the damage becomes life-threatening.

In the formation of a venous thrombus, primarily protein structures of the liquid part of the blood (plasma) and, to a lesser extent, cells are involved. There are no cholesterol plaques in the venous bed, but there is a valvular apparatus. If we consider a person who is in a vertical position, then the blood in the venous bed of the legs flows from the bottom up, that is, against gravity. In order to prevent blood from flowing back down, there are valves inside the veins, which are semi-lunar petals, outgrowths of the vein wall. In addition, the blood in the venous system has a lower blood flow velocity, a decrease in which can lead to venous blood stasis.

Macroscopically, venous blood stasis can manifest itself in the form of such a phenomenon as sludge – settling and crowding of heavier blood fractions (cells and protein macromolecules). A fairly clear example of the formation of sludge can be called pouring compote into a glass. If you stir the compote with a spoon, its elements will be evenly distributed over the glass, and then settle to the bottom. Blood behaves in approximately the same way during venous blood stagnation, only the elements of the “compote” inside it are very small. Of course, the sweetness of the blood itself is not of great importance with normal thrombolytic activity of the plasma (the system that dissolves blood clots) and without other provoking factors. However, this may create prerequisites for venous thrombosis.

If we talk about the venous system of the legs, the heart (right sections), the diaphragm and the lungs are responsible for pumping blood, as well as a very important mechanism called the muscle pump of the leg. The fact is that inside the gastrocnemius muscle there are branched venous reservoirs, represented by the sural (sural) and popliteal veins. When the leg muscles contract (for example, while walking), venous blood from these veins is pumped into the femoral vein and then into the veins of the small pelvis, providing an acceleration of blood flow in the venous system and preventing stagnation. When we are in an upright position or sit still, the venous “pump” of the lower leg does not work, which means that blood stasis increases. All these features create the prerequisites for the formation of thrombosis.

Deep and superficial veins: a big difference

If we consider venous thrombosis from the point of view of nosology, then there are diseases that contribute to its development. There are some nuances in the development of deep and superficial vein thrombosis that distinguish them from each other. The most common cause of superficial (saphenous) vein thrombosis is, of course, varicose veins.

Varicose veins are an enlargement of the subcutaneous veins with a violation of their function, because the valves inside them do not work, which makes it impossible to raise blood up to the heart. As the valves stop closing in a vein that is overloaded with blood, a reverse (top-down) flow of blood, or “reflux” is formed.

Varicothrombophlebitis of the great saphenous vein

In addition, venous blood in such a vein is constantly in a state of sludge. Overstretching of the vein leads to changes in the inner wall (intima) of the diseased venous vessel, which should prevent the adhesion (sticking) of blood cells and protein molecules to it. Under unfavorable circumstances, a pronounced blood sludge occurs in such a vein, a decrease in blood flow velocity, as well as inflammation in the vein wall.

External impact (strike, bruise, insect bite), as well as overheating, loss of fluid in hot or hypothermia in the cold season, viral and other diseases with a rise in temperature can lead to the formation of thrombosis and its subsequent growth along the diseased varicose vein. This is how thrombophlebitis is formed – superficial vein thrombosis, which occurs with inflammatory skin changes, pain and redness over varicose veins.

Thrombophlebitis of the trunk of the great saphenous vein of the left leg

According to statistics, in 70-90% of cases, thrombophlebitis occurs against the background of varicose veins. Thrombosis of healthy (non-varicose) superficial veins accounts for about 10% of cases: as a rule, these are patients with cancer or thrombophilias (genetic defects in proteins involved in thrombosis). Thus, the most common cause of superficial vein thrombosis is varicose veins.

Superficial (saphenous) vein thrombosis is dangerous if it spreads to the deep venous system. However, the presence of a thrombus in the superficial veins forces doctors to apply more active treatment tactics. If thrombosis is localized in the tributaries of the main saphenous vein, it is possible to use local anti-inflammatory drugs and compression therapy (knee socks or stockings, depending on the localization of thrombosis).

Thrombus migration from the superficial vein to the deep vein (with the development of embolism)

Thrombus migration from the superficial vein to the deep venous system is a dangerous complication of superficial vein thrombosis with possible pulmonary vein thromboembolism. Watch the video on my channel https://www.youtube.com/watch?v=uAKyIHKliXU

Most phlebologists recommend using anticoagulants. For the treatment of superficial vein thrombosis in the territory of the Russian Federation, only one drug is certified – this is parnaparin sodium ( fluxum ). Using another drug to treat saphenous vein thrombosis is legally wrong.

Parnaparin sodium (fluxum)

Surgical techniques are mainly aimed at preventing the spread of a blood clot to the deep veins (ascending path). This is a crossectomy (ligation and crossing of the great saphenous vein and the vessels flowing into it on the leg) in the area of ​​the saphenofemoral (the confluence of the great saphenous vein with the deep femoral) or sapheno-popliteal fistulas, as well as endovascular methods (radiofrequency or laser crossectomy). Usually, both methods are supplemented with anticoagulants (drugs that thin the blood).

Removal of a thrombus from a vein during surgical treatment of varicose veins (miniphlebectomy). Raskin V.V. operates.

Removed venous thrombus

How was the removal of the thrombus during endovenous laser ablation? Watch the video on my youtube channel. https://www.youtube.com/watch?v=7kqs7iNP_XQ

In 2019, the latest recommendations of the Association of Phlebologists of Russia on the diagnosis and treatment of thrombophlebitis of the superficial veins of the extremities were published. The text of the recommendations can be viewed here https://www.mediasphera.ru/issues/flebologiya/2019/2/119976976201

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As for the deep veins, which provide the main outflow of blood, the most common causes of thrombosis are tumors, prolonged immobilization of the limb during injuries or bed rest during operations, the use of hormonal contraceptives, thrombophilia, as well as deep thrombosis combined with varicose thrombosis veins. Thrombosis and blockage of these veins leads to a violation of the outflow of fluid from the leg and the appearance of edema below the thrombosis zone. The patient experiences pain due to overstretching of muscle and skin tissues as a result of edema, where many different tissue receptors are located, including pain receptors. The edematous limb often has a bluish tint.

Ultrasound angioscanning (ultrasound of the veins of the legs) is performed to confirm or exclude venous thrombosis. If after the study it is not possible to exclude thrombosis, there is the possibility of laboratory confirmation – a blood test for d-dimer.

Acute (recent) deep vein thrombosis is treated in several ways. The simplest is the appointment of therapeutic doses of anticoagulants (drugs that block the formation of thrombus masses). These include subcutaneous or intravenous heparin , low molecular weight heparins ( clexane, fraxiparin ), fondaparinux ( arixtra ), as well as tablet forms – vitamin K antagonists ( warfarin, syncumar ) and new oral anticoagulants (NOACs) – rivaroxaban ( xarelto ), apixaban ( eliquis ), dabigatran ( pradaxa ). All of them block the synthesis of a new blood clot. The dissolution of the thrombus occurs due to the predominance of its own thrombolytic activity of the blood plasma with blocked thrombus-forming activity. Thus, these drugs do not dissolve the clot on their own. In this regard, despite the rather high efficacy of drugs, in some cases, thrombosis can persist and even continue to grow. Dissolution (recanalization) of thrombosis takes from several weeks to several months.

Rivaroxaban (xarelto) is an effective drug for the treatment of deep vein thrombosis

Another technique is thrombolysis, that is, the administration of drugs that act directly on the thrombus. The technique is effective and allows you to dissolve fresh blood clots within a few hours or days. At the same time, due to the complete blockage of the thrombogenic system, severe uncontrolled bleeding is possible, up to death in patients with a high risk of bleeding (for peptic ulcers of the digestive tract and some other diseases). Therefore, thrombolytic therapy is carried out under the constant supervision of qualified doctors in a hospital or intensive care unit.

A modern method of administering thrombolytics is catheter-guided thrombolysis (CUT), when the drug is injected locally directly into the thrombus (through a specially installed catheter). This allows you to optimize the dosage and inject the drug directly into the affected area. Unfortunately, in Russia only a small number of clinics have the opportunity to use this technology. There are also endovascular (intravenous) systems for the mechanical removal of a thrombus from a vein, but their implementation has not yet become widespread.

The more time has passed since the moment of thrombosis, the less effective conservative methods of treatment show. This is explained by the structural reorganization of the thrombosis zone: the replacement of the thrombus by a dense connective tissue – a scar, sometimes with the inclusion of calcium. In this situation, the only way to help the patient is through surgery. The modern technique consists in balloon angioplasty and stenting of the affected occluded (closed) vein. However, unresolved questions remain about the need for anticoagulant therapy after stenting, the choice of stents and drugs, as well as possible reflux (reverse blood flow) in the stented areas, since the valve apparatus of the vein in this area is irreversibly damaged.

Post-thrombotic disease in a patient with long-standing thrombosis of the popliteal vein of the left leg

Summing up all of the above, I note that any thrombosis requires proper examination and treatment. In the case of arterial thrombosis, therapy should be carried out as urgently as possible in specialized vascular departments. With venous – depends on the duration of the onset of symptoms and the localization of the thrombus. In a number of situations with venous thrombosis (saphenous vein thrombosis, distal deep vein thrombosis without edematous syndrome), outpatient treatment (at home) or endovascular outpatient surgery is possible. Proximal thromboses (at the level of the femoral vein, iliac vein, or inferior vena cava) require hospitalization and observation to prevent thrombus separation and thromboembolism (blockage) of the pulmonary artery.

Review of the surgeon-phlebologist Raskin Vladimir Vyacheslavovich

I would like to express my gratitude to the surgeon-phlebologist Raskin Vladimir Vyacheslavovich for the skillfully performed operation, his high professionalism in his work, for his “golden hands” and polite attitude towards the patient. In March 2017, I underwent an operation – endovenous laser obliteration with Varadi miniphlebectomy on both legs. The quality of work is impeccable, which provided me with a quick recovery. Dr. Raskin Vladimir Vyacheslavovich is a responsible and attentive doctor. Good luck and good luck to you! Thanks again!

Olenin A.V.
Ramenskoye, Moscow region
July 5, 2017

1. How to make an appointment with a phlebologist? Where is the reception?

Make an appointment with a vascular surgeon, phlebologist, Ph. D. Raskin Vladimir Vyacheslavovich, please call +7 (495) 641-62-77. You can read information about the doctor here https://phlebo.center/o-doktore

Reception is held in the city phlebological center at the address: Kolomenskaya metro station, Yakornaya st., 7, building 1 Please see the address and location map here https ://phlebo.center/kontakty

2. What should I take with me to an appointment with a phlebologist?

Take your medical documentation with you (extracts, doctors’ consultations on your disease or concomitant pathology), as well as a list of drugs that you take constantly

3. Do I need to do an ultrasound of the veins before seeing a phlebologist?

Ultrasound of the veins is not necessary before seeing a phlebologist, because our phlebologists themselves have an expert class in ultrasound diagnostics of leg veins. However, if you have already done such studies, take the results with you so that the doctor can assess the dynamics of the disease.

4. What tests should be done before seeing a phlebologist?

It is not necessary to take tests before the initial appointment. However, if you have already done such studies, take the results with you so that the doctor can assess the dynamics of the disease.

5. How much does it cost to see a phlebologist?

Make an appointment with a vascular surgeon, phlebologist, Ph.D. Raskin Vladimir Vyacheslavovich and to find out the latest information on the cost of admission and procedures, please call +7 (495) 641-62-77. Here you can also get acquainted with the prices for diagnostics and treatment https://phlebo.center/price

Vladimir RASKIN, phlebologist, candidate of medical sciences.

How to defeat lower limb vein thrombosis using modern techniquesPhlebological Center “Antireflux”

The formation of blood clots in the veins of the lower extremities is a common deviation that is not always possible to diagnose in the early stages due to poor symptoms. Few people pay attention to slight swelling and heaviness, and in the meantime, the disease progresses and begins to threaten not only health, but also the life of the patient. Thrombosis of the veins of the lower limb can lead to death. If earlier treatment was reduced to extensive surgical intervention, now everything has become much easier. Older people with overweight, women taking oral contraceptives, as well as citizens who have suffered injuries are at risk of blood clots . Thrombosis can also form as a result of a major operation, in pregnant women and bedridden patients.

How to diagnose and treat lower limb vein thrombosis in a hospital

If you belong to a risk group, it is necessary to periodically perform ultrasound of the veins and dopplerography of the vessels. If a blood clot is detected, the doctor will prescribe a number of additional examinations that will help clarify the size and exact location of the blood clot. When there is a possibility of separation, immediate hospitalization is necessary in a specialized center where experienced specialists work.

Thrombosis of the veins of the lower extremities is visible to the naked eye of a doctor . If you find a prominent swollen vein on your leg, which swells and hurts especially strongly at the end of the evening, immediately contact a phlebologist for further recommendations and treatment. Thrombi that are well attached to the venous walls will be dissolved by taking anticoagulants. Weekly blood control is required.

It is more difficult to diagnose thrombosis of veins located in the muscle layer. That is why the treatment of deep vein thrombosis of the lower extremities is more complex and lengthy, and sometimes surgery is necessary. For prevention, you need to undergo ultrasound diagnostics at least once a year in order to start therapy in a timely manner.

Treatment to prevent thromboembolism

If we consider all methods of treatment, then drug therapy is most often prescribed. Therefore, there is no need to worry when going to the doctor for examination. In some cases, hospitalization is necessary. Treatment of lower extremity thrombosis can sometimes be done at home. Trust the choice of therapy to the doctor. Even aspirin should not be taken on its own with thrombosis. Do not rush to turn to traditional medicine, you can harm yourself and lubricate the symptoms.

Treatment of deep vein thrombosis of the lower extremities requires a special approach, since it is in these places that large floating thrombi are formed, threatening embolism. Usually, either drug therapy is prescribed in a hospital setting, or minimally invasive techniques for removing blood clots are used. If possible, a cava filter is installed, resembling an hourglass or an umbrella, in order to fix the thrombus and conduct further intensive anticoagulant therapy.

In rare cases, when the clot is located in places that are difficult to reach for devices, conventional surgery is recommended. Recovery in the treatment of deep vein thrombosis of the lower extremities in this way occurs within 2-4 weeks.