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Keratosis pilaris after pregnancy: Keratosis Pilaris: Little Bumps, Big Annoyance – Skin and Beauty Center


Keratosis Pilaris: Little Bumps, Big Annoyance – Skin and Beauty Center

It’s like having goose bumps when you’re not cold. Keratosis pilaris turns soft, smooth skin into sandpaper. This common skin condition most often causes arm bumps, but it can also created bumps on the upper thighs, buttocks, and face.

Doctors don’t know why some people develop these arm bumps and others don’t, but there are ways to treat keratosis pilaris.

What Causes It, Who Gets It

Normally, skin cells imperceptibly flake off on their own. But that doesn’t happen for people with keratosis pilaris. For those with this skin condition, the skin protein keratin plugs the hair follicles and causes tiny white or red bumps that may be itchy. The bumps aren’t painful, but can make the skin feel dry and rough.

Although it can happen at any age, this is a skin condition that generally affects the young. Babies may have it, particularly on their cheeks, but it’s most common in teenagers on the upper arms. It can also appear on the upper thighs or buttocks.

Keratosis pilaris tends to decrease or disappear by age 30. In the meantime, it can become worse during the winter when humidity is low and the skin is dry. The arm bumps may also get worse during pregnancy or after giving birth.

Keratosis pilaris is not rare: Nearly 4 in 10 people have it. People who already have dry skin or eczema are at higher risk for developing it. It also runs in families.

Keratosis Pilaris Treatment Tips

These skin bumps are harmless, so you don’t have to treat them if they don’t bother you. However, if you’d like to get rid of keratosis pilaris, try these steps:

  • Start by moisturizing. Moisturize your skin several times a day, especially after bathing. You can try plain petroleum jelly, such as Vaseline, or creams like Acid Mantle or Complex 15. If these moisturizers don’t do the trick, try an over-the-counter medicated cream that contains urea, lactic acid, glycolic acid, salicylic acid, or vitamin D.
  • Consider a prescription. A dermatologist may advise using a chemical peel that can strip the extra skin cells or a topical retinoid (a vitamin A derivative) that you can get with a prescription, such as tretinoin (Retin-A, Renova). If arm bumps and other bumps caused by keratosis pilaris are very red and appear to be infected, your doctor may prescribe antibiotics.
  • Take a hot bath and exfoliate. Rubbing your skin with a washcloth, brush, or exfoliating body sponge after an occasional long, hot bath can help dislodge the plugs in your hair follicles and make your skin smoother. The rest of the time, avoid drying out your skin by taking short, warm showers or baths and using mild soaps.
  • Get help from a humidifier. Using a home humidifier when the humidity is low can help prevent keratosis pilaris from getting worse in the winter.
  • Shave with a gel or cream. If keratosis pilaris affects areas you typically shave, make sure to shave after you’ve bathed, and let the shaving gel or cream sit on your skin for a few minutes before you start shaving. Getting hairs soft will help minimize irritation.

When it comes to treating keratosis pilaris, persistence is key. Keeping your skin moisturized and seeing your doctor for a prescription product can help you keep it under control. In the meantime, know that it usually goes away during adulthood, so the future should look brighter … and smoother.

Learn more in the Everyday Health Skin and Beauty Center.

Keratosis pilaris – NHS

Keratosis pilaris is a very common harmless condition where small bumps appear on your skin. It can last for a long time, but there are treatments that may improve your skin.

Check if you have keratosis pilaris

Symptoms of keratosis pilaris may include:

Dry rough skin.


Small, painless bumps on your skin.


You usually get patches of small bumps on your arms, thighs or bottom, but they can appear in other places.

The bumps are usually the colour of your skin. They can also be red on white skin or brownish-black on darker skin.

The skin can sometimes feel itchy, and may be better in summer and worse in winter.

Keratosis pilaris can affect people of any age. If your child has a rash and you’re not sure it’s keratosis pilaris, see more about rashes in babies and children.

Things you can do yourself to help

Most people with keratosis pilaris have it for years, and it may eventually clear up by itself.

Until it does, there are things you can do to help improve the appearance of your skin.


  • moisturise your skin – ask a pharmacist what’s most suitable for you

  • use mild and unperfumed soaps and bathing products

  • gently scrub your skin with a washcloth or exfoliating mitt

  • have cool or lukewarm showers and baths

  • pat your skin dry instead of rubbing it after washing


  • do not use perfumed soaps or bathing products that can dry out your skin

  • do not use harsh scrubs on your skin – this can make it worse

  • do not have hot baths or showers

  • do not scratch, pick or rub your skin

A pharmacist can help with keratosis pilaris

Speak to a pharmacist if:

  • things you try yourself are not helping your keratosis pilaris and the condition is bothering you
  • your skin becomes itchy or inflamed
  • you’re not sure if you need to see a GP

They can recommend creams or lotion to help your skin. They can also tell you whether you need to see a GP.

Specialist treatments

Specialist treatments for keratosis pilaris include steroid creams and laser treatments.

These types of treatments are not usually available on the NHS and can be expensive to pay for privately. While they may help relieve symptoms in some cases, they do not cure keratosis pilaris, so symptoms can come back after a course of treatment.


Keratosis pilaris happens when your hair follicles become blocked with a build-up of keratin, a substance found in skin, hair and nails.

Nobody knows exactly why keratin builds up, but the condition is thought to run in families. So if your parents have it, you may get it, too.

Keratosis pilaris is not infectious, so you cannot spread or catch it.

Page last reviewed: 25 June 2021
Next review due: 25 June 2024

Gluten And Keratosis Pilaris AKA Chicken Skin Rash

Chicken skin is a term commonly used for the skin condition keratosis pilaris. According to “Dr. Google,” this rash on the backs of arms, cheeks, and thighs is made worse by eating gluten. Can cutting gluten from the diet help? Is there truth to this claim, or is it another internet myth?

This rash is seen in up to 50% of the world’s population. These tiny bumps can be cosmetically unappealing but don’t cause other symptoms or harm. They can be emotionally distressing for some, especially at times of flare-ups.

My patients often ask me what causes these bumps? Is it a gluten rash? Are there other diet changes that help?  How can I get rid of it?.

Keratosis pilaris ( aka chicken skin rash or KP) occurs due to overproduction or build-up of keratin, a protective protein found on the skin. The bumps can be surrounded by redness, a sign of inflammation seen when they are looked at under a microscope.

Keratosis pilaris occurs more often in people with eczema or dry skin and worsens in cold or dry weather. It is associated with other inflammatory conditions such as allergies and asthma.

Keratosis pilaris affects 50-80% of adolescents and up to 40% of adults. It may flare during puberty and decrease with age.

There is a strong genetic component so it often runs in families. Symptoms typically come and go throughout the year. Picking at the bumps will only make them more visible and increase scarring and discoloration.

Although it is known to have a genetic factor, scientists do not know the true cause of keratosis pilaris. It is seen more commonly in inflammatory conditions such as asthma and allergies.

It associated with dry skin, vitamin A and essential fatty acid deficiency. Flares and remissions may occur with hormonal changes such as pregnancy.

There are no studies indicating a direct correlation between gluten and keratosis pilaris ( chicken skin). However, it can be caused by vitamin A deficiency or essential fatty acid deficiency, both of which can occur with gastrointestinal absorption problems.

If you have celiac disease or gluten sensitivity you are at risk of poor absorption. In theory, this could increase your odds of having keratosis pilaris due to these nutrient deficiencies.

Aside from an association with low vitamin A or low essential fatty acids, there are few studies showing a correlation between diet and keratosis pilaris. Increasing essential fatty acid intake by consuming more coldwater fish such as sardines, mackerel, and salmon may lessen the rash. Walnuts are a great vegan source of essential fatty acids.

There are many people who do say their rash is affected by other diet changes but no studies to back up those claims. Some people say cutting out dairy or sugar has improved their rash. In general, consuming an anti-inflammatory diet may be of benefit and is not harmful but there are no studies to say an anti-inflammatory diet improves keratosis pilaris.

Hydration of the skin is an important part of treatment for keratosis pilaris. Mild cases can be treated with over-the-counter lubricating creams. There is a laundry list of dermatologic therapies such as alpha-hydroxy creams, retinoic acid therapies, and steroids. Dermatologists may prescribe personalized therapies that can be formulated.

Further specific medications are beyond the scope of this article but can be found here. For more severe cases, laser therapy and dermabrasion may be used.

The most common alternative treatment for a ” chicken skin rash ” are omega-3 fatty acids ( fish oil supplements). In addition to providing the essential fatty acids that may be lacking, these supplements also have a strong anti-inflammatory effect.

Dietary manipulations such as gluten-free or dairy-free, anti-inflammatory, sugar-free, Paleo, or various autoimmune diet protocols are often implemented. A variety of ‘gut healing’ regimens are also prescribed, but as with most therapies surrounding keratosis pilaris, there is no evidence to support or refute them.

Topical therapies may include coconut oil, olive oil or vitamin A capsules. Finally, exfoliants such as baking soda, oatmeal, or sugar or salt scrubs are sometimes helpful in reducing keratin build-up. Most people do not seek treatment for keratosis pilaris unless it is cosmetically significant, so home therapies are a popular approach.

3 Treatments for Keratosis Pilaris – Cleveland Clinic

It’s finally tank-top season, but your upper arms are looking like a plucked chicken. What’s with those tiny pimple-like bumps that make your skin feel like sandpaper?

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Let us introduce you to keratosis pilaris or KP. “It’s totally harmless and super common,” says dermatologist Alok Vij, MD.

That doesn’t make the unsightly speckles any less annoying. Dr. Vij shares the easiest ways to banish the bumps.

What is keratosis pilaris?

KP occurs when keratin, a protein in skin, builds up around the hair follicles. The result is rough, raised bumps that can look skin-colored, white, brown or red. It’s particularly common on the upper arms, but can show up in other spots, too, including the thighs or face.

The bumps are more common in kids and teenagers, who often outgrow them. But for some individuals, KP has staying power. People with eczema are also more likely to experience the (not-so-joyful) joys of KP.

Treating keratosis pilaris

If KP is bugging you, your first instinct might be to grab a loofah and start scrubbing. But step away from the exfoliators.

Abrasive scrubs and body brushes can trigger your skin to make even more of the protein that’s causing KP.

So how can you get the smooth skin of your dreams? “It’s always better to moisturize than to try to scratch the bumps away,” he says.

Dr. Vij recommends these three ingredients, all available in over-the-counter products found in most drugstores.

  1. Ceramides. These proteins help skin hold onto moisture. Moisturizers with ceramides can help reduce roughness in people with KP. (The ingredient also helps fight eczema, so it’s a good choice if you’re dealing with both skin problems.)
  2. Urea. Urea is a naturally occurring molecule that helps dissolve the keratin protein gumming up the follicles. It’s in many drugstore moisturizers. Your doctor can also prescribe creams with a higher concentration of urea.
  3. Ammonium lactate. This ingredient also helps dissolve the buildup that causes KP’s telltale bumps and redness. It’s a little bit sticky, Dr. Vij warns, so it’s not everyone’s first choice. But if you don’t have luck with ceramides or urea, it’s worth a shot.

Tips for tackling KP

Getting rid of KP takes some patience. Dr. Vij recommends:

  • Use one of the recommended moisturizers daily for four to six weeks.
  • If you don’t notice improvement, try one of the other types.
  • If THAT doesn’t work, you might have to combine two ingredients or see a dermatologist for a prescription-strength option.
  • Once you find a cream that works for you, you’ll likely have to keep using it to keep the bumps at bay.

Some people with KP are more bothered by the redness than the roughness — especially those with fair skin, and those who have KP on the face. In those cases, laser treatments done in a dermatologist’s office can help fade the red, Dr. Vij says.

You may need some trial and error to get rid of the chicken skin. But if KP is making you crazy, it’s nice to know that a trip to the drugstore might be all you need to send those little bumps packing.  

Keratosis Pilaris Condition, Treatments, and Pictures for Parents – Overview


Information for

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Images of Keratosis Pilaris


Keratosis pilaris is a very common harmless skin condition appearing as small, whitish bumps on the upper arms and thighs, especially of children and young adults.

Individual lesions of keratosis pilaris begin when a hair follicle becomes plugged with keratin, a protein found in skin, hair, and nails.

Who’s at risk?

Keratosis pilaris can affect people of any age, any race, and either sex. It is more common in females.

Keratosis pilaris usually starts in early childhood (by age 10) and can worsen during puberty. However, it frequently improves or even goes away by early adulthood.

Keratosis pilaris can affect 50–80% of teenagers and up to 40% of adults. Many people have a family history of keratosis pilaris. A large number of individuals with ichthyosis vulgaris (an inherited skin condition characterized by very dry, very scaly skin) also report having keratosis pilaris.

Signs and Symptoms

The most common locations for keratosis pilaris include the following:

  • Backs of the upper arms
  • Fronts and sides of the thighs
  • Buttocks
  • Cheeks

Tiny (1–2 mm) white to gray bumps occur, centered in the hair follicle. Sometimes, a thin, red ring may surround the white bump, indicating inflammation. The bumps all look very similar to one another, and they are evenly spaced on the skin surface.

Rarely, people with keratosis pilaris may complain of mild itching.

Keratosis pilaris tends to improve in warmer, more humid weather, and it may worsen in colder, drier weather.

Self-Care Guidelines

There is no cure for keratosis pilaris, though its appearance can be improved. It is often helpful to keep the skin moist (hydrated) and to use mild, fragrance-free cleansers, with daily applications of moisturizer.

Creams and ointments are better moisturizers than lotions, and they work best when applied just after bathing, while the skin is still moist. The following over-the-counter products may be helpful:

  • Preparations containing alpha-hydroxy acids such as glycolic acid or lactic acid
  • Creams containing urea
  • Over-the-counter cortisone cream (if the areas are itchy)

Do not try to scrub the bumps away with a pumice stone or similar harsh material; these approaches may irritate the skin and worsen the condition. Similarly, try to discourage your child from scratching or picking at the bumps, as these actions can lead to bacterial infections or scarring.

When to Seek Medical Care

Keratosis pilaris is not a serious medical condition and has no health implications. However, if self-care measures are not improving the appearance of the skin and it continues to bother your child, see your child’s doctor or a dermatologist who may recommend more aggressive treatments.

Treatments Your Physician May Prescribe

Keratosis pilaris usually improves with time. However, it is generally considered to be a long-lasting (chronic) skin condition. Treatments are aimed at controlling the rough bumps, not curing them. Keratosis pilaris bumps will come back if therapy is stopped.

To treat the bumps of keratosis pilaris, the doctor may recommend a topical cream or lotion containing:

  • Prescription-strength alpha- or beta-hydroxy acids (glycolic acid, lactic acid, salicylic acid)
  • Prescription-strength urea
  • A retinoid such as tretinoin or tazarotene
  • High concentrations of propylene glycol

For keratosis pilaris that is itchy or inflamed, the doctor may recommend the short-term use of a topical corticosteroid to reduce the inflammation.

Trusted Links

Clinical Information and Differential Diagnosis of Keratosis Pilaris


Bolognia, Jean L., ed. Dermatology, pp.205-206. New York: Mosby, 2003.

Freedberg, Irwin M., ed. Fitzpatrick’s Dermatology in General Medicine. 6th ed, pp.486, 714, 1216. New York: McGraw-Hill, 2003.

Symptoms, Causes, Treatment, and Prevention

What Is Keratosis Pilaris?


Keratosis pilaris is a common, harmless skin condition that causes small, hard bumps that may make your skin feel like sandpaper. You may have heard it called “chicken skin.”

The bumps are often light-colored. They usually appear on your upper arms, thighs, and buttocks, sometimes with redness or swelling. They can also show up on your face, but that’s less common.

Many children and teens get it, and it usually disappears as they get older.

Symptoms of Keratosis Pilaris

Signs of keratosis pilaris include:

Except for some possible itching, keratosis pilaris doesn’t hurt and doesn’t get worse. 


Causes of Keratosis Pilaris

Keratosis pilaris is caused by a buildup of keratin, the protein that protects skin from infections and other harmful things. The buildup forms a plug that blocks the opening of a hair follicle, but doctors don’t know what triggers the buildup.

If you have dry skin, you’re more likely to have keratosis pilaris. It’s usually worse in the winter months, when there’s less moisture in the air, and then may clear up in the summer.

It often affects people with certain skin conditions, including eczema (also called atopic dermatitis).

Diagnosis of Keratosis Pilaris

Your doctor can diagnose keratosis pilaris by looking at your skin. You don’t need to be tested for it.

Keratosis Pilaris Treatment

There’s no cure for keratosis pilaris. But moisturizing lotions or creams may help your skin look and feel better. A variety of these are available over the counter, but you’ll need a prescription for stronger versions.

Two types of products that go directly on the affected skin often improve keratosis pilaris. You’ll need to use them daily for several weeks before you’ll see a change.  

Topical exfoliants remove dead skin cells from the surface of your skin. These include creams that contain alpha-hydroxy acid, lactic acid, salicylic acid, or urea.

The acids may cause redness or a slight burning, so they aren’t recommended for young children.

Topical retinoids, related to vitamin A, help prevent hair follicles from getting plugged. These include products with the ingredients tretinoin (Atralin, Avita, Renova, and Retin-A) and tazarotene (Avage, Tazorac). But topical retinoids may irritate your skin or cause redness or peeling.

Women who are pregnant, nursing, or may become pregnant should avoid topical retinoids.

Laser treatment — aiming a laser onto the skin — is sometimes used to treat severe redness and inflammation. It isn’t a cure, but it may provide some relief when creams and lotions aren’t enough. You might need several sessions for this treatment to work.

Home remedies for keratosis pilaris

Keep your skin moist to lessen its effects.

Some simple things can help keep your skin comfortable:

  • Don’t scratch at the bumps or rub your skin roughly.

  • Use warm water rather than hot for bathing and showering.

  • Limit your time in the water.

  • Try soap that has added oil or fat.

  • Use thick moisturizers generously on the skin.

  • Add moisture to the air in your home with a humidifier.

  • Don’t wear tight clothes (friction can irritate your skin).

A Guide to Managing Keratosis Pilaris This Season

As a 20-year sufferer of keratosis pilaris, the notion of Jergens lotion ad-worthy skin—impossibly smooth, virtually poreless, and preternaturally glowing—has always evaded me. Over time, I’ve grown accustomed to the tell-tale permanent goosebumps on the back of my arms and legs (you might know the term “strawberry legs”), as well as accepted the fact that there is no cure for this common skin condition. That being said, I’ve also relished doing what I can do to manage it, and know that while nothing can banish the bumps forever (at least not yet), there are plenty of ways to lessen symptoms thanks to a spate of innovative products and treatments.

As temperatures rise, hemlines shorten, and limbs are seeing the light of day for the first time in a while, here experts break down what KP is, along with the best ways to treat it this summer, from gentle exfoliation to supercharged moisture.

Get to Know Keratosis Pilaris

Keratosis Pilaris, or KP, is an incredibly common skin condition that affects 40% of adults. It typically manifests as small, hard bumps, usually surrounded by red areas of skin, on the upper arms, legs, or buttocks. In simplest terms, KP is a blockage of the hair follicles. “The skin just creates a buildup of a protein called keratin—the same building block for hair, skin and nails—and the keratin creates a plug that blocks the hair follicle, which leads to the little bumps,” explains explains New York City dermatologist Rachel Nazarian, M.D., of Schweiger Dermatology. “The plug can also trigger inflammation in the skin, which is what causes the redness around each hair follicle.” Despite how prevalent KP is, and that it tends to run in families, it’s unknown why some people experience it and others don’t, though Nazarian says it’s often found with people who have very sensitive skin, or alongside conditions such as eczema or atopic dermatitis.

Lean Into the Heat of Summer

While KP is usually asymptomatic, it is often worse in the winter and less noticeable in the summer. “Arid climates and dry skin worsens KP as skin cells are more adherent and shed less frequently when they are dry,” explains Blair Murphy-Rose, MD, FAAD, dermatologist, and Clinical Assistant Professor at New York Presbyterian Hospital-Weill Cornell Medical Center. “Higher humidity levels and sunlight are believed to help with controlling KP.” While this serves as an impetus to hit the beach and catch some rays this summer, layering on a sunscreen with an SPF of at least 30 is essential to protect yourself against skin cancer and premature aging.

Exfoliate Regularly—But With Kid Gloves

Let’s start with what not to do. “The most common mistake I see my patients making is trying to ‘scrub’ the bumps off, or using a rough loofah to exfoliate the bumps away,” explains Nazarian. “Although this technique will work temporarily by dislodging the keratin plugs, rough exfoliation will inflame the skin and hair more, ultimately causing increasing redness and making the condition even more noticeable.” While physical exfoliators can often be too abrasive on KP, there are specially formulated, finely-milled body scrubs, such as First Aid Beauty KP Bump Eraser, that can be effective. “If you are someone that loves a good body scrub, just make sure to scrub gently and always moisturize immediately after,” says Murphy-Rose, who recommends patting skin dry with a towel after bathing and applying a rich moisturizer while still damp.

First Aid Beauty KP Bump Eraser Body Scrub with 10% AHA

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Soft Services Smoothing Set

Goshi Exfoliating Shower Towel

But while there’s instant gratification in a scrub that leaves skin smooth, experts agree that chemical exfoliation, which uses acids to gently dissolve away dead skin, is the best strategy for treating keratosis pilaris. According to Murphy-Rose, options for chemical exfoliation include topical lotions, creams, or serums that contain glycolic acid and lactic acid (AHAs), salicylic acid (a BHA), gluconolactone (a PHA), and urea. Offering the ultimate hybrid between physical and chemical is Soft Services, a new and impossibly stylish body-care brand founded by Glossier alums Annie Kreighbaum and Rebecca Zhou. Two of its debut products are the Buffing Bar, an exfoliating brick featuring ultrafine microcrystals and hydrating shea butter, and Smoothing Solution, a gel exfoliant laced with lactic acid, urea, and soothing centella.

In-office at the dermatologist, chemical peels, ranging from mild daily peels to stronger in-office monthly or bi-weekly peels, can offer more dramatic results. Additionally, some patients get great results against KP by using retinol or a prescription-strength retinoid. “These vitamin A derivatives help to regulate skin cell turnover and encourage appropriate shedding of skin cells,” says Murphy-Rose.

Stacked Skincare TCA Lactic & Glycolic Body Peel

REN Clean Skincare AHA Smart Renewal Body Serum

SkinCeuticals Body Retexturing Treatment

Obagi KeraPhine Body Smoothing Lotion

Glytone Exfoliating Body Lotion

Replenix Gly-Sal 10-2 Clarifying Pads

Moisturize, Moisturize, Moisturize

For controlling KP, moisture is key—especially when the temperatures drop and air becomes more arid. “Anything that dries out skin can make KP more noticeable,” explains Nazarian. “This includes arid seasonal changes and many patients find that their KP flares in the winter and fall, when humidity drops and moisture is pulled out of the skin more easily. ” When looking for lotions and creams to slather on, a combination of humectant, emollient, and occlusive ingredients make for the most effective moisturizers. “Look for ingredients like ceramides, shea butter, petrolatum, dimethicone, glycerin, and hyaluronic acid,” says Murphy-Rose, adding that anti-inflammatory properties, like aloe and niacinamide, can be helpful in reducing discoloration in reddish bumps. In the spirit of a practicality, Murphy-Rose advises her KP patients to use a moisturizer that also contains exfoliating ingredients, such as the Obagi KeraPhine Body Smoothing Lotion or Soft Services Carea Cream, so it can function as a 2-in-1. “The hardest part about KP treatment is sticking to a strict twice daily regimen, so having an ‘all-in-one’ product is always a plus,” she says.

It can’t be emphasized enough: Sticking to a dedicated regimen of exfoliating and moisturizing is key for keeping KP continuously under control. Otherwise, it will return to normal. “The continuous use of the creams will help smooth skin, followed by the eventual fading of the surrounding redness, but once treatment is discontinued the bumps reappear,” says Nazarian. “I remind my patients not to get discouraged—many people out-grow the condition and it typically lessens as they get older.”

Gold Bond Rough & Bumpy Daily Skin Therapy

Vichy Ideal Body Lotion Serum-Milk with Hyaluronic Acid

CeraVe SA Cream For Rough & Bumpy Skin

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90,000 Follicular hyperkeratosis of the skin: symptoms, treatment, diagnosis of the disease

Follicular hyperkeratosis is called skin pathology, which consists in the formation of nodules of the keratinized epidermis at the mouths of the hair follicles. The skin on the affected areas becomes dry and rough, covered with numerous reddish nodules that resemble goose bumps. Most often, the disease affects the elbows and knees, gluteal zones, outer thighs.It does not cause any deterioration in well-being, although it does cause some discomfort due to the feeling of dry skin and dissatisfaction with its appearance.

Types of disease

There are two types of follicular hyperkeratosis of the skin.

  • Type I develops with a lack of vitamin A. It is characterized by increased dryness of the skin on the elbows, knees, extensor surfaces of the limbs and buttocks and the appearance of spiny nodules in the mouths of the hair follicles.
  • Type II develops with a lack of vitamin C. Its difference is the dark color of the nodules that clog the mouths of the follicles, since they consist of blood or pigment. The outer thighs and abdomen are most commonly affected.

The disease can be congenital or acquired.


The main symptom of follicular hyperkeratosis is “goose bumps”, which is accompanied by increased dryness and roughness of the skin on the affected areas.Keratinous nodules look like a small reddish or yellowish rash that appears on the arms and thighs, knees, buttocks, and less often on other parts of the body. They are located at the base of the hair follicles with the formation of a small red rim around each horny element. Nodules and plaques do not exceed the size of a match head and can persist on the affected skin for many years. In some cases, manifestations of follicular hyperkeratosis appear on the face or on the scalp.The generalized form of the disease is characterized by extensive skin lesions on the trunk and limbs.

Do you have symptoms of follicular hyperkeratosis?

Only a doctor can accurately diagnose the disease.
Do not delay the consultation – call

+7 (495) 775-73-60

The reasons for the development of pathology

Normally, epithelial cells appear in the basal layer of the skin, filling with keratin and hardening as they grow.As new cells appear, the old ones peel off almost imperceptibly and leave the skin surface. The cell renewal cycle takes about two days.

When it is violated, old cells do not have time to fully mature and keratinize, therefore, when displaced by young cells, they do not peel off, but accumulate, increasing the thickness of the stratum corneum. In this case, the lumens of the hair follicles may be closed by a thickened layer of horny cells. The follicles become clogged, which limits the growth of new hairs to the subcutaneous space.This is how follicular hyperkeratosis develops with acne or nodules on the affected skin.

In some cases, the disease is hereditary, but there are a number of reasons that trigger this mechanism. Among them:

  • hormonal imbalance inherent in adolescence and adolescence, or taking hormonal drugs;
  • stress, increased emotional stress or high physical exertion;
  • malnutrition, hypovitaminosis or vitamin deficiency;
  • winter period and associated skin irritation with layers of clothing.

The causes of acquired follicular hyperkeratosis, as a rule, are:

  • lack of vitamins A, C, B, E and K;
  • thyroid dysfunction;
  • systemic diseases of the body;
  • severe infectious diseases – tuberculosis, syphilis, HIV, etc . ;
  • disorders of the development of connective tissue;
  • traumatic external influences – chemical burns, radioactive radiation;
  • tight and uncomfortable synthetic fiber clothing.

Follicular hyperkeratosis most often develops in children, adolescents and adolescence, but it also occurs in adults.

Diagnostic Methods

Follicular hyperkeratosis, as a rule, does not require special diagnostic studies. For an experienced dermatologist, the results of an external examination and a patient survey are usually sufficient to determine the nature of the disease. In adolescents, keratinization nodules can sometimes be confused with acne, but close examination reveals dry and rough skin, in contrast to the soft structure inherent in acne.In some cases, it may be necessary for differential diagnosis if the manifestations of follicular hyperkeratosis are similar to:

  • lichen red;
  • pityriasis versicolor;
  • Darier’s disease;
  • follicular psoriasis;
  • lenticular keratosis.

In these cases, a histological examination of the nodules is performed to study the nature of the skin lesions and determine the cause of their occurrence.

Pathology treatment

Since the disease is chronic, the treatment of follicular hyperkeratosis of the skin continues for a long time. Often, the patient has to struggle with its manifestations throughout his life. Therapy is usually aimed at improving the condition of the skin, preventing skin infections and inflammation. Quite often, a disease that began in childhood or adolescence, with the onset of maturity, independently regresses, and its manifestations become less noticeable.However, the need for medical attention remains.

An integrated approach to the treatment of follicular hyperkeratosis can significantly improve the condition of the skin. This requires:

  • to normalize the diet by including a large amount of vegetables and fruits, fatty fish, lean meat;
  • take medications prescribed by a dermatologist – retinoids, vitamin complexes;
  • regularly use external agents containing corticosteroids, salicylic acid, AHA acids, urea;
  • in the presence of a fungal infection, use antifungal drugs;
  • attend physiotherapy sessions – quartzing, laser therapy, photodynamic therapy and other prescribed procedures;
  • Take warm baths with salt, baking soda or starch.

Compliance with all the doctor’s recommendations allows you to achieve long-term stable remission, getting rid of the manifestations of the disease for a long time.

Diagnostics and treatment of follicular hyperkeratosis in Moscow

Clinic “Medicine” offers effective treatment of follicular hyperkeratosis in children, adolescents and adult patients. Take advantage of the services of qualified dermatologists, as well as the widest possibilities of the latest diagnostic and treatment equipment, to restore the health and smoothness of the skin.Make an appointment with a specialist online on our website or by phone. We are waiting for you any day of the week at a convenient time for you.

Questions and answers

Which doctor treats follicular hyperkeratosis?

For the diagnosis of follicular hyperkeratosis and subsequent treatment, consult a highly qualified dermatologist.

What is the danger of follicular hyperkeratosis?

Follicular hyperkeratosis itself is not a dangerous disease, but in some cases it is complicated by pustular skin infections, irritation and inflammation of the affected skin areas, and the development of eczema. In some, very rare cases, the degeneration of the affected tissues into cells of a malignant tumor is possible.

Follicular hyperkeratosis: what helps from folk remedies?

Some traditional medicine recipes help soften skin, reduce breakouts, and relieve irritation. Most effective:

  • aloe leaves kept in the refrigerator for several days, applied to the affected area overnight;
  • gruel from grated raw potatoes or raw beets;
  • bee propolis.

Atopic Dermatitis – Diagnosis, Symptoms and Treatment in EMC

Atopic dermatitis (or neurodermatitis) is one of the most common skin diseases. This is an inflammatory skin lesion of an allergic nature, which causes many different factors – allergens entering the body with food, by contact (in contact with the skin) or by inhalation. The main manifestations are skin rashes, most often in places of large folds, face, neck and, most importantly, itching. Beginning, as a rule, in childhood, the disease becomes chronic and proceeds with periods of exacerbations.

Since its discovery, the disease had more than a hundred designations, until in 1923 Coca and Cooke proposed the term “atopia” (from Greek – strangeness, unusualness) to define the state of hypersensitivity in hay fever, asthma, “atopic eczema”, which later in 1933 was renamed Wize and Sulzberger “atopic dermatitis”. Since then, this name has been generally accepted.

Atopic dermatitis is a multifactorial inflammatory skin disease. Its development is influenced by heredity, disorders in the immune system and an unfavorable environment.

Causes of the disease

The primary onset of symptoms of the disease occurs under the influence of various external and internal factors, mainly in children of the first years of life. Atopic dermatitis can recur, which often leads to psychological problems in the process of personality formation and a decrease in the quality of life in adulthood.

One of the main reasons for the development of atopic dermatitis is a genetic predisposition. It has been proven that more than 20 genes can participate in the development of allergic diseases.

According to research, atopic dermatitis develops in 80% of children whose parents have this disease. More than 50% of children inherit this disease if only one parent is sick. Moreover, if atopic dermatitis is diagnosed in the mother, the risk increases by 1.5 times.

Stages of development of the disease

Most often, atopic dermatitis manifests itself in childhood and has different stages of the course; symptoms may vary depending on age.

There is no single generally accepted classification of atopic dermatitis, but there is a working version, according to which 4 stages of the disease are distinguished.

Initial stage . As a rule, it develops in children with increased vulnerability of the skin and mucous membranes, instability of water-salt metabolism, a tendency to allergic reactions and a decrease in resistance to various infections. Distinctive symptoms of the stage: hyperemia, swelling and peeling of the skin on the cheeks. With timely and correct therapy, it is completely cured.Symptoms do not go away on their own, with improper and untimely treatment, an exacerbation of symptoms or a transition of the disease to a more severe stage is possible.

Stage of pronounced skin changes, or stage of progression . It almost always includes two phases: acute and chronic.

Stage of remission . Disappearance or significant reduction of symptoms. The duration of the stage is from several weeks to 5-7 years. In difficult cases, the disease proceeds without remission and persists throughout life.

Stage of clinical recovery . According to the data of clinical diagnostics, the manifestations of the disease are absent for 3-7 years or more.

Forms of atopic dermatitis

Each form is characterized by the presence of itching of varying intensity.

Infant form (from birth to 2 years) . Redness and small blisters appear on the skin, from which a bloody fluid is released when pressed. The liquid, when dry, turns into yellowish brown crusts.Itching worse at night. As a result of scratching, marks and cracks appear on the skin. Symptoms most often appear on the face, can be on the arms and legs (in the elbow and popliteal folds), buttocks.

Children’s uniform (3-7 years old) . Redness, swelling, crusts appear on the skin, the integrity of the skin is disrupted, the skin becomes thicker, the skin pattern is more pronounced. Nodules, plaques and erosion are formed. Cracks in the palms, fingers and feet are very painful.

Teenage uniform (8 years and older) .Red plaques with vague edges appear on the skin, pronounced dryness of the skin, many itchy cracks. Most often, the disease is localized on the flexor surfaces of the arms and legs, wrists, dorsum of the feet and palms.

Prevalence of atopic dermatitis

Limited . The disease manifests itself only in the neck, wrist, elbow and popliteal folds, palms and the back of the feet. The rest of the skin remains unchanged.Moderate itching.

Common . The disease occupies more than 5% of the body surface. The rash spreads to the limbs, chest, and back. The rest of the skin becomes earthy. The itching becomes more intense.

Diffuse . The entire surface of the skin is affected. Itching is pronounced and intense.

Severity of current

Estimated on the basis of the intensity of skin rashes, the extent of the process, the size of the lymph nodes, etc.

Light current . Differs in mild hyperemia, fluid secretion (exudation), peeling, single rashes, mild itching. Exacerbations occur 1-2 times a year.

Moderate course . Fluid secretion increases, multiple lesions appear. The itching becomes more intense. Exacerbations occur 3-4 times a year.

Heavy current . There are multiple extensive lesions, deep cracks, erosion.Itching intensifies, becomes persistent.


Clinical manifestations are characterized by erythematous, exudative and lichenoid eruptions, which are accompanied by intense itching. Atopic dermatitis is often associated with abnormalities in skin barrier function, sensitization to allergens, and recurrent skin infections. Dysbacteriosis of the skin microbiota may also play a key role in the development of atopic dermatitis. It has been scientifically proven that atopic dermatitis is a skin symptom of a systemic disorder and often appears as the first step in the so-called “atopic march” that causes bronchial asthma, food allergies and allergic rhinitis.

The main symptoms of atopic dermatitis

  • ichthyosis, xerosis, dry skin
  • palms hyperlinearity
  • darkening of the skin of the eye sockets
  • Hertog’s sign (lack or absence of hair from the outer part of the eyebrows)
  • Denier-Morgan fold (longitudinal fold of the lower eyelid)
  • persistent white dermographism
  • lichen white, hair lichen
  • follicular keratosis
  • folds on the front of the neck

Additional symptoms:

  • pallor
  • low hairline
  • delayed reaction to acetylcholine
  • linear grooves at fingertips
  • keratoconus or cataract

How is atopic dermatitis diagnosed?

Diagnosis of atopic dermatitis begins with a mandatory visit to a dermatologist-allergist. Advanced allergy diagnostics includes laboratory blood tests for allergens, diagnostics of food intolerances, molecular diagnostics, and prick tests.

Molecular diagnostics is the most modern method that allows you to identify the causal factors of the disease as accurately and quickly as possible, even in the most difficult cases, when other types of analyzes turn out to be uninformative.

Basic diagnostic criteria:

  • Itchy skin
  • Skin rashes: in children under 2 years of age – on the face and on the bends of the elbows and knees, in older children and adults – thickening of the skin, increased pattern, pigmentation and scratching in the bend of the limbs
  • Chronic course with the possibility of relapses
  • Family history of atopic diseases
  • The disease first appeared before the age of 2 years

Additional indicators:

  • Seasonality of exacerbations (progression in autumn and winter, regression in summer)
  • Exacerbation of the disease in the presence of provoking factors (allergens, food, stress, etc. )etc.)
  • Increase in total and specific IgE in the blood
  • Increase in the level of eosinophils (a subspecies of granulocytic leukocytes) in the blood
  • Hyperlinearity of palms (increased number of folds) and soles
  • Follicular hyperkeratosis (“goose bumps”) on the shoulders, forearms, elbows
  • The onset of itching with increased sweating
  • Dry skin (xerosis)
  • White dermographism
  • Tendency to skin infections
  • Localization of the pathological process on the hands and feet
  • Nipple eczema
  • Recurrent conjunctivitis
  • Hyperpigmentation of the skin around the eyes
  • Folds on the front of the neck
  • Denier-Morgan symptom
  • Cheilitis (inflammation of the red border and mucous membrane of the lips)

To establish a diagnosis, a combination of 3 main and at least 3 additional criteria is required.

Diagnostics of the effectiveness of therapy

Assessment of severity

  • EASI: index of the area and severity of eczema (for the doctor). Assessment of prevalence in 4 separate areas (head and neck, trunk, upper limbs, lower limbs).
  • POEM: Eczema severity questionnaire (for patients). The patient himself assesses the severity and intensity of symptoms for the last 7 days, answering the questionnaire.

Assessment of the patient’s quality of life

  • Dermatological Quality of Life Index (DLQI).The patient assesses the effect of the disease on the severity of symptoms, sensations, daily activity, leisure, work / study performance, personal relationships and treatment within a short period of time (1 week)
  • WPAI Questionnaire: SHP. Evaluation of the impact of atopic dermatitis on productivity during the last 7 days. How the course of the disease affected the ability to work and perform daily activities.

Complications of the disease

Atopic dermatitis is often complicated by the development of a secondary infection (bacterial, fungal or viral).The most common infectious complication is the appearance of a secondary bacterial infection in the form of strepto- and / or staphyloderma. Pyococcal (purulent) complications appear in the form of various forms of purulent skin lesions: ostiofolliculitis, folliculitis, vulgar or streptococcal impetigo, boils.

Various fungal infections (dermatophytes, yeasts, molds and other types of fungi) also often complicate the course of atopic dermatitis, negatively affect the effectiveness of treatment.The presence of a fungal infection can change the symptoms of atopic dermatitis: there are foci with clear scalloped and raised edges, cheilitis often recurs, lesions behind the ear, inguinal folds, nail bed, and genitals are possible.

Patients with atopic dermatitis are more likely to suffer from a viral infection (herpes simplex virus, human papillomavirus). Herpes infection can provoke the development of a rare and serious complication – Kaposi’s herpetic eczema. With Kaposi’s eczema, widespread rashes appear, severe itching, fever rises, and a purulent infection quickly joins.In some cases, the central nervous system and eyes are affected, and sepsis develops.

Enlargement of lymph nodes in the cervical, axillary, inguinal and femoral regions may be associated with exacerbation of atopic dermatitis. This condition goes away on its own, or after adequate treatment.

Ophthalmic complications of atopic dermatitis – recurrent conjunctivitis accompanied by itching. Chronic conjunctivitis can progress to ectropion (eversion of the eyelid) and cause watery eyes.

Treatment methods

Effective treatment of atopic dermatitis is impossible without a systematic approach, which includes:

  • Elimination measures: prevention of contact with irritants, including in food, and household allergens.
  • Regardless of the severity of the disease, if necessary, treatment is supplemented with antihistamines, antibacterial, antiviral and antimycotic agents.
  • Emollients and moisturizers to restore impaired skin barrier function.It is now known that the addition of a ceramide-dominated emollient to standard therapy results in both clinical improvement and a decrease in water loss through the skin and improved stratum corneum integrity. These funds are recommended for use as maintenance therapy during remission.
  • Topical glucocorticosteroids are the basis of anti-inflammatory treatment, showing high efficacy in the fight against acute and chronic inflammation of the skin in limited lesions.Due to concerns about the potential side effects associated with chronic use, these drugs are not used for maintenance therapy.
  • Calcineurin inhibitors are absolutely safe for face and eyelid skin lesions. Several studies of pimecrolimus cream have shown that the use of the drug in the earliest stages of the disease leads to a significant decrease in the need for “rescue” therapy with glucocorticosteroids.
  • In case of a moderate course, the use of phototherapy is relevant.It acts on inflammatory cells (neutrophils, eosinophils, macrophages, Langerhans cells) and alters the production of cytokines, and also has a persistent antibacterial effect. Moreover, UV phototherapy can have a normalizing effect on the immune status.
  • In a severe course of atopic dermatitis, in addition to topical agents, treatment includes the use of systemic glucocorticosteroids in short courses and cyclosporine. In 55% of cases, the positive effect occurs after 6-8 weeks of use.Continuous therapy is not recommended for more than 1–2 years because cyclosporine has potential toxicity.
  • Biological therapy in the treatment of atopic dermatitis.

Dupilumab is the world’s first monoclonal antibody-based treatment for atopic dermatitis. This is a class of drugs that are highly selective for the key components of the pathological process. Antibodies have the ability to accurately bind to the antigen due to special antigen-binding sites that have high specificity for it.For drugs based on antibodies, this determines their selectivity for a particular target. New drugs work where previous drugs fail. For this reason, they can be more effective or used in cases where the disease has proven resistant to traditional drugs.

Also recently, omalizumab was found to be the most effective drug in the treatment of allergic asthma and allergic rhinitis. Thus, it can potentially neutralize the action of immunoglobulin in atopic dermatitis.

Prevention of atopic dermatitis

In atopic dermatitis, itchy skin can be provoked even by completely harmless factors, for example, clothing and moisture. That is why it is recommended to wear natural fabrics and avoid strenuous physical activity. Washing powders, even in small amounts remaining on linen and clothes, can irritate the skin, therefore it is recommended to use hypoallergenic soap-based detergents, adding a repeated rinse cycle of the linen.You should also exclude products containing fragrances and preservatives, and try new cosmetic products on a small area of ​​the skin before use.

It is necessary to cleanse and moisturize the skin, because with atopic dermatitis, due to disturbances in lipid metabolism of the skin, its dryness increases. Violation of the protective function can lead to the development of secondary bacterial, viral and fungal infections. That is why the skin with atopic dermatitis requires special care: shower cleansers (“soap without soap”), as well as bath oils; after washing, without rubbing the skin, you must immediately apply a moisturizer.Moisturizers and balms can be applied several times a day.

Primary prevention

Primary prevention is aimed at preventing the development of atopic dermatitis and should be carried out during pregnancy. It is known that the disease is inherited, and if both parents have it, the probability of getting sick in the unborn child is 60 – 80%.

It is recommended to exclude highly allergenic foods such as chocolate, citrus fruits, honey, nuts, etc. from the diet of a pregnant woman.On the other hand, the diet should be varied, one-sided carbohydrate diet should be avoided. Timely treatment of preeclampsia is important, which significantly increase the permeability of the “placenta-fetus” barrier and contribute to allergization. It is recommended to limit the medication load, as many medications can cause allergies. Elimination of overload at work and occupational hazards during pregnancy.

Prophylaxis after childbirth is no less important, where breastfeeding plays a very important role, since breast milk is maximally adapted to the needs of the newborn and does not contain foreign proteins that may cause allergies.

For a child with a predisposition to atopic dermatitis, drug treatment should be prescribed only for clearly justified indications, since drugs can act as allergens that stimulate the release of immunoglobulin.

Secondary prevention

Performed when a child is diagnosed with atopic dermatitis and is intended to reduce the number of exacerbations and improve the quality of life.

Exacerbations of atopic dermatitis can cause:

  • house dust mites;
  • mold formed on the ground of domestic plants and in damp areas;
  • components of cosmetics or detergents;
  • animal hair, etc.

In this case, regular wet cleaning (including with the use of washing vacuum cleaners), frequent change of bed linen, treatment of tiled walls with antifungal solutions, use of clothes made from natural fabrics (except for wool) will help.

You can make an appointment with an EMC dermatologist by phone: +7 (495) 933 66 55.

90,000 Hair loss in women

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Hair loss in women – Consider a variety of causes of hair loss in women:

Hormonal disorders can act as a cause of profuse hair loss in women.So, the problem may lie either in the imbalance of hormones, or in the excess content of the male hormone testosterone. This happens with endocrine diseases, at the beginning of sexual activity, during pregnancy, menopause in women, etc. Hair loss is observed when prescribing and canceling contraceptives, as well as during pregnancy, and especially, in the postpartum period.

Aggressive environmental factors negatively affect all body systems and can cause hair loss. It considers polluted air in big cities, increased radiation levels, the content of negative and even hazardous substances in precipitation, bad habits (smoking and drinking alcohol), exposure to low and high temperatures (no headgear during frost and heat).Mechanical reasons – curling, using a hair dryer, and others.
Hair loss often occurs due to reduced immunity. When the body is weakened by disease. With anemia, hypovitaminosis, accompanying various diseases.

Diagnosis in women differs little from diagnostic methods in men, with the exception of only consulting a gynecologist-endocrinologist.

Forms of hair loss in women

All causes of hair loss in women can be divided into two groups: telogen hair loss (when hair falls out gradually) and anogenous alopecia (that is, sudden hair loss).

Anagenic alopecia

This is a sudden loss of hair, most often caused by exposure to strong chemicals or drugs, as well as radiation. It is this type of baldness that occurs during chemotherapy. As a rule, with mild exposure, the activity of the hair follicles is subsequently restored. There is no specific treatment for this type of hair loss. Hair growth is restored after the cessation of harmful effects, under the influence of stimulating drugs.

Telogen hair loss

Telogen baldness can occur in both acute and chronic forms. The difference is that acute baldness lasts up to six months, and then, after therapy, hair growth is restored. Chronic telogen hair loss can last for more than six months and sometimes does not go away even for several years. With this type of alopecia, complete baldness does not occur.

Treatment: Finding and eliminating the leading cause of alopecia is the mainstay of treatment.

Also carried out:

  • Head massage, laser treatment, electrical stimulation and other physiotherapeutic methods such as microcurrent therapy.
  • Reception of vitamin complexes, balanced nutrition. Preparations and products with a high content of zinc, selenium, fatty acids, amino acids, B vitamins
  • Use of external anti-hair loss agents containing vitamins, arginine, herbal complexes to improve microcirculation and nutrition (only as an auxiliary therapy).
  • Plasma therapy, injections of bioactive drugs

Clinical Study Follicular Keratosis: Salkera Softening Foam – Clinical Research Register



Inclusion criteria: – The subject is willing and able to give an informed message. – The subject is willing and able to participate in the study on an outpatient basis and is willing to meet the requirements of the study. – Subject is 18 years of age or older.- Subject has 2 of 4 limbs. – For the evaluated limb, the subject has at least moderate severity (≥ 3) on the Investigator’s on-site assessment of CP severity. – For each limb assessed, the subject scores at least 6 points on the Investigator’s Assessment of Erythema, Roughness, and Scaling. – If the subject is a woman of childbearing age, the subject will test negative in urine. pregnancy test at screening (0 week). – If female, subject will be either postmenopausal for> 2 years, surgically sterilized.(hysterectomy or bilateral uterine ligation) or the use of one of the forms over fertility (abstinence, oral contraceptives, estrogen patches, contraceptives on implants, injectable contraception, IUD, diaphragm, condom, sponge, spermicide or partner vasectomy). Female subjects must use contraception for 1 month after completing their studies. Exclusion Criterion: – Subject of a clinically significant, unstable, or poorly controlled health condition as identified by the investigators / sub-investigators.- Subjects have an active skin infection, atopic dermatitis, or any other skin disease that would interfere with the clinical assessment of CP. – Subject is allergic to any ingredient in the study drug. – A subject who has used any of the following treatments for CP lesions within the past two weeks: topical corticosteroids, tretinoin, tazarotene, adapalene, salicylic acid, alpha hydroxy acid, lotion with urea and / or ammonium lactate. – Subject who has been treated with UVB therapy in the past two weeks.- A subject who has received systemic antibiotics, a steroid, tacrolimus, tretinoin, isotretinoin and / or PUVA within the past 4 weeks. – Female subjects, pregnant (positive pregnancy test), breastfeeding, or considering becoming pregnant during the study period. – A subject who is currently participating in another clinical study or has completed a clinical trial within the past 4 weeks.



Minimum age:

18 years old

Maximum age:

N / A

Healthy volunteers:

Accepts healthy volunteers

follicular keratosis ((- 86 responses to Babyblog

Not fun…Nasty byaka ((For life. Well, maybe until the age of adolescence, if you’re lucky. You know, these small pimples don’t seem to itch and are not particularly noticeable, but a rough cheek, a leg, a pen to the touch …

Small pimples with a white head appeared, such dry ones first on the cheeks. The cheeks still turned red, then turned pale, did not itch (thank God!). from 5. At first, the pediatricians thought that it was “irritation from saliva”, then “like diathesis”, then “everything will pass” … Now the baby is a year and 2 months old. pimples on the legs, on the shoulders and on the cheeks.Finally we got to a dermatologist. The diagnosis is follicular keratosis (dermatitis). How to treat? Not cured! How to live, and what should you do? Review the child’s diet. Remove: juices, sweets, fresh vegetables, ready-made cereals, mashed potatoes. You can: only boiled porridge with your own hand without sugar; 2 apples, 2 pears, 2 banana, 2 slices of bread – per week; drink water, black and green tea is not sweet; cottage cheese is not sweet without additives; stewed vegetables only their own, chicken, meat, eggs only their own. Soups, potatoes, pasta, biscuits, a couple of dryers.In general, sweet in the form of sweets, chocolate, I didn’t give him anyway, but the baby certainly used juice, Heinz porridge and mashed potatoes, sweet agusha. Further it is allowed to use a scrub. Make masks with kefir, white clay. A moisturizer is a must. Then I went to the pediatrician (not my own). All this was advised by Enterosgel – to remove toxins from the body. Enterosgel is a good thing, already tested and works for me personally. Vooot …. In general, someone passes this byaka in childhood, someone in a transitional age, and someone does not go through their whole life.

Dear ones, if someone came across such a bad name, then which creams helped better, and which means it is better not to waste time at all and not to expose the child. ?

It is also called “goose bumps”. I dug for general information and according to the dermatologist this is what it is. (I am citing excerpts from the Internet, because I myself cannot describe this byaka so neatly, I found it on another forum) “My son was diagnosed with the same diagnosis, but in America. In the Russian Internet, I did not find the normal information presented on this topic, but there are many articles on the English-speaking Internet devoted to this problem.I can briefly tell you what I learned for myself: Firstly, this disease is hereditary, it is present in almost 50 percent of the world’s population. By the way, I also have such pimples, my mother had it in childhood, but passed with age. Such pimples occur because the hair follicles are clogged with keratin, a protein that is part of the upper protective layer of the skin. Nails and hair are also made of keratin. Keratin layers and forms pimples, often popularly called goose bumps.Actually, this is the problem, that these scales do not peel off, like other people, but grow. I understand that this is precisely due to heredity, the normal mechanism of this process is somehow disrupted. In fact, these are not pimples, they can blush, but this is more often associated with some kind of irritants. Usually in winter, all this is exacerbated, in summer, especially after exposure to the sun, the skin condition improves. Often, the condition also worsens if the skin is dry, this just often happens in winter, so it is imperative to use not oily, but a moisturizer, preferably immediately after water procedures.There is an opinion that most often people with this problem have a deficiency of vitamins A and E. In principle, children have enough moisture, nothing else can be done. Adults are advised to use scrubs and immediately moisturizer, as well as creams with acids, they help dissolve these plugs, they also use retinol, etc. … “

P.S. I do not assert anything and I do not call for anything! I collect opinions and information only for myself!

90,000 Electrolysis during pregnancy is possible or not

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The miracle of the birth of a new life entails an incredible amount of change for a woman. Her body is involved in a process leading to fluctuations in hormones and, often, to an increase in the latter. This results not only in mood swings, but also in the increased growth of unwanted hair.

The main culprit for this problem is the androgen. Vegetation selects a wide variety of areas of the body, including the face, abdomen, and chest area. And the acceleration of metabolism and blood circulation enhances the nutrition of the roots, thereby more strongly stimulating their growth.

Photo from www.shutterstock.com

Is it possible to do electrolysis “in position”?

It’s no secret that expectant mothers sometimes feel less attractive, and unexpected additional “hair problems” only intensify these feelings.

And here a desire arises to eliminate this annoying effect. In addition, it is likely that the girl performed electrolysis treatment before pregnancy and does not want to stop the course of procedures.

And it is also possible that it is necessary to remove only a few shoots from the face area. There is a legitimate question about the safety of electrolysis.

This topic is still controversial. The opinions of experts are sometimes radically different from each other. Let’s try to collect points of view and summarize.

A little theory

Before we start interviewing professionals, let’s refresh an extremely important point that will play a huge role in understanding the issue.The point is that there is little confusion about electrical methods for removing vegetation.

Electrolysis combines several types, which are based on two types of current:

  • Thermolysis or diathermy is a low intensity alternating current that is converted into heat and burns out the follicle.
  • Another method is based on galvanic constant electricity, which affects the chemical composition of the liquid surrounding the root – electrolysis.
  • The combination of these methods is called blends.
  • An increase in the intensity of thermolysis with a decrease in exposure time is known as flash.

It’s easy to get confused about these complex terms. But we don’t really need it.

It is important to understand one single difference: thermolysis acts locally within the removed element, while electroplating, on the other hand, travels throughout the body, from the apparatus to the opposite charge in contact with the body, and vice versa.

Thermolysis works like moxibustion, and the direct current acts on body fluids.

As you know, the fetus is located in space, surrounded by amniotic fluid. Consequently, the galvanic method is highly likely to pass through these formations. Remembering this important detail, it will be easier for us to participate in the debate.

Let us refer to foreign sources

Let’s find out the opinion of our European and American colleagues.

What is advised in the USA:

Some American colleagues rightly recall the fact that for a period of more than 100 years, no negative effect on pregnancy of the discussed method of hair removal has been officially discovered.

Thereby, believing that this is a completely acceptable way with the condition of observing certain points: obtaining permission from the attending physician and following a special procedure technique.

For example, knowing that electrolysis travels from one charge to another within the body, electrologists recommend shortening the distance of such a journey as much as possible.

For example, if the work is in the area of ​​the left hand, then the return electrode should be placed under the left hand. That is, the area of ​​the abdomen and small pelvis is completely turned off from contact.

Also, these experts believe that during the first 6 months it is permissible to treat the bikini area if the return electrode is located under the thigh of the treated side. The last trimester is a contraindication for sessions on the chest and abdomen.

According to another point of view, electrolysis is completely safe in any area during the period of fetal wear.

British Electrolysis Association estimates:

Specialists of the British Electrolysis Association adhere to the following vision of the situation.

“Usually, pregnancy is not a contraindication to thermolysis (diathermy). There is no risk to the mother or fetus. Avoid treating the area of ​​the chest and abdomen, but do it only because of the stretching of the skin of these areas. ”

They are inclined to believe that harm to the fetus cannot be caused, since the current does not pass through the body.However, it is best to refrain from blending and electrolysis methods during the entire period.

However, if you have accidentally done the procedure, please do not worry. These procedures can be carried out without any consequences, especially if the work is done on the face. According to their statistics, a large number of women, not knowing about their situation, attended procedures without negative consequences.

According to another part of professionals, electrolysis cannot be performed in the first 3 months due to the likely stress.In the following months, only thermolysis is possible. Other methods are strictly contraindicated.

Genetic Consultant Laurie Wolfie:

Genetic consultant Laurie Wolfie believes that although the galvanized appearance is undesirable, the risk of any consequences is close to a minimum. With regard to diathermy, the doctor is absolutely calm and recommends this method without hesitation.

The specialist advises against working in the nipple area due to the likely soreness of subsequent breastfeeding.Also, the geneticist warns against carrying out the procedure if the hair appears only in position, since after the birth of the baby, the vegetation can disappear on its own.

Director of the DCLH Center in New York:

Director of the Center for Dermatology, Cosmetology and Laser Surgery in New York, Dr. David Bank, also joins the idea of ​​the safety of the procedure.

In contrast to the above opinions, no scientific research has been carried out on the effect of electrolysis on pregnancy, therefore, this is still an insufficiently studied issue.

And this is the basis of the conviction of another part of specialists about the extreme undesirability of carrying out this method throughout the entire period.

Russian practice:

The opinions of Russian practitioners agree on the unanimous point of view that pregnancy is a direct and categorical contraindication for the electrical technique due to its adverse effects on the health and bearing of a child.

We are not talking about the difference between direct and alternating current, electrolysis and thermolysis.The answer is no!

Basically, doctors believe that the stress of the procedure and its negative consequences are the main cause of possible complications. Objectively, such a decision has one justification: it is better not to take risks.


Summing up, we made sure that this issue is tactically resolved in different ways in Russia and abroad. Of course, colleagues from other countries rely on theory, statistics and a long history.

For the most part, foreign doctors consider thermolysis to be a completely safe method, and galvanization is an undesirable, but acceptable method, subject to certain precautions.

Domestic doctors flatly refuse to even talk about the admissibility of this procedure during the entire period of gestation, without examining whether it is electrolysis or thermolysis.

Having understood the theory of the question, it is easy to assume that this procedure is technically quite acceptable.

Given the lack of knowledge of the issue, the lack of information, as well as the huge responsibility not only for the life of a woman, but also for an unborn child, it makes sense to join the opinion of Russian professionals and refuse electrolysis during this period.

As mentioned above, hormones, tastes, and preferences change at this time. Any impact that used to be normal can cause allergies, stress and unwanted consequences during this delicate period.

In our center, pregnancy is a contraindication not only for electrolysis, but also for other methods of hair removal. The most important thing for us is taking care of your health and the health of your baby.

And it is better to return to the issues of hair removal after childbirth and even breastfeeding.


Medically written and reviewed by: Julia Nicholson, dermatologist, physiotherapist
Published by: Olesya Smagina, Assistant Director of the Universe of Beauty Hair Removal Centers

»Treatment of keratoma

Keratoma is a benign tumor that does not occur on the outer part of the skin.At the beginning of the course of the disease, it appears in the form of a darker colored spot on the skin, which later begins to coarse and peel off. The disease usually occurs on exposed areas of the skin that are exposed to sunlight. Under the influence of ultraviolet radiation, the growth and keratinization of the outer skin scales occurs. The proliferation of keratoma is often focal in nature, the tumor grows in size and similar formations appear in adjacent areas of the skin. The disease is not contagious and is not transmitted by contact with a healthy person.Keratosis usually occurs in people over 35 who are often exposed to the sun. Keratoma very rarely degenerates into a malignant formation, however, self-diagnosis of keratosis is difficult. To accurately determine the type of tumor, you must consult a specialist.

Types of keratomas

The following types of keratosis are most common:
1. Senile keratosis : occurs in the elderly over 60 years of age in the form of spots on the skin that differ in color from healthy tissue.Usually appears on the face, neck and hands and tends to develop focal.
2. Seborrheic keratosis (seborrheic keratoma): Appears on areas of the skin that are covered with hair in the form of a scaly dark patch. It is most often found on the hands, feet or head, the parts most exposed to sunlight. In addition to aesthetic discomfort, a keratoma can itch and cause other discomfort. When parts of keratinized tissue fall off, micro-wounds appear, into which infection can get.3. Horny keratosis : A tumor that protrudes a few millimeters above the skin and is clearly defined. The swelling is harder and rougher to the touch than normal skin. The size of the tumor can be about 3 – 5 mm in diameter. This type of keratosis can develop into a malignant formation over time.
4. Follicular keratosis : small nodules (1 – 2 mm) that develop under the scalp. Basically, this type of keratoma occurs on the head.5. Actinic (or solar) keratosis : the most dangerous type of keratosis, but, fortunately, the rarest. This type is a precancerous skin disorder. It manifests itself, like ordinary keratosis, in the form of gray spots on open areas of the skin covered with keratinized crusts, but has a malignant tumor structure. If untreated, this type of keratosis degenerates into squamous cell skin cancer.

Removal of keratomas

Methods of treatment of keratosis and removal of keratomas:
1.Surgical method: the neoplasm is mechanically removed with surgical instruments. After removal, the wound is treated and sutured. This method is effective, albeit traumatic. After it, scars often remain, and they are undesirable on visible areas of the skin. In state medicine, such formations are made mainly by a surgical method.
2. Cryodestruction: the damaged tissue is frozen with liquid nitrogen, after which the tumor dies. To achieve the result, several procedures are required with an interval of two to three days.It is not possible to completely remove the tumor in one visit to the clinic, which is the main disadvantage of this method.
3. Laser Removal : The surgical laser is the most modern method of minimally invasive surgery. The laser beam vaporizes the tumor tissue at a predetermined depth without damaging healthy skin. The laser has the ability to coagulate blood vessels, which minimizes bleeding and the possibility of subsequent inflammation. The operation is performed under local anesthesia, which makes it painless.The wound after laser removal of the keratoma heals in about 5-6 days, and the redness completely disappears within two weeks. The entire operation takes about 30 minutes and is done on an outpatient basis in 1 visit to the surgeon.

Removal with keratomas by laser

The following advantages of laser keratosis treatment are highlighted:

  • laser keratoma removal is a completely non-contact method because the tumor tissue is evaporated by a beam of light.
  • the wound after the operation is completely treated with a laser beam, which excludes the possibility of re-inflammation.
  • operation is performed on an outpatient basis in 1 visit to the clinic.
  • the patient’s recovery period after surgery is very short.
  • there are no side effects when removing a keratoma with a laser.

Removal of keratoma in Moscow

For the treatment of keratosis in Moscow, we suggest you use the services of our clinic for the following reasons:

  1. Latest equipment: In our clinic we use only the latest surgical or cosmetic lasers.Operations are carried out in a modern operating room that fully meets all approved standards.
  2. Experienced doctors: Our center has been in existence for over 15 years. During this time, we have already performed thousands of successful operations. Our doctors, dermatologists – surgeons have vast practical experience in removing neoplasms on the skin.
  3. Removal of keratomas without pain: surgery to remove the tumor is performed under local anesthesia, which is selected individually for each case.
  4. Examination before removal: before removal, we will definitely make sure that the tumor is not malignant. This ensures that there are no consequences after removal.
  5. Transparency of work: before removal, the attending physician will fully tell you how the operation will proceed and discuss the price with you.
  6. Saving time: we value your time very much, therefore we will coordinate the appointment time with you in advance so that you do not have to wait. On average, the operation to remove a keratoma takes 20 to 30 minutes.

Get free detailed consultation by phone (495) 434-34-44 or (495) 670-20-28 from 8 to 20 hours daily.

Our addresses : Moscow, Metro Yugo-Zapadnaya or Belyaevo / Konkovo, st. 26 Baku commissars, 6; Moscow, metro station “Proletarskaya” st. Talalikhina, 2/1, bldg. 5.

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Came to consult about a tumor on the hand. It turned out to be a keratoma. The doctor diagnosed it and immediately suggested removing it with a laser.It took about 15 minutes in total, I waited longer in the queue. the previous patient was delayed. I liked the surgeon Elena Valerievna very much, she was attentive and professional. I recommend everyone to contact her.

Peter (Moscow, 2014)

I brought my dad to the clinic with a keratoma on his arm, when it already began to look completely unpresentable. The tumor was removed in one visit with a laser. It took 30 minutes in total. The clinic is small, but I liked the environment very much. Everything is fast and professional.Prices are strictly according to the price list. Nothing extra.

Zhanna (Moscow, June 2013)

We went to the Clinic with a problem of multiple keratomas (in my mother). We made an appointment, got to an excellent specialist (Sergey Gennadievich). After a competent consultation, they remained for removal, the doctor did everything very quickly and efficiently.